Figures
Defects in zebrafish embryos
Expression of mutant Shp2 with a mutation that was identified in human Noonan Syndrome results in a range of defects in zebrafish embryos, including shortening of the body axis (top three embryos) compared to a control non-injected embryo (bottom) (see Jopling et al., e225).
Image Credit: Photograph provided by Chris Jopling, Hubrecht Institute.
Citation: (2007) PLoS Genetics Issue Image | Vol. 3(12) December 2007. PLoS Genet 3(12): ev03.i12. https://doi.org/10.1371/image.pgen.v03.i12
Published: December 28, 2007
Copyright: © 2007 Jopling et al.. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Expression of mutant Shp2 with a mutation that was identified in human Noonan Syndrome results in a range of defects in zebrafish embryos, including shortening of the body axis (top three embryos) compared to a control non-injected embryo (bottom) (see Jopling et al., e225).
Image Credit: Photograph provided by Chris Jopling, Hubrecht Institute.