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Co-existing TP53 and ARID1A mutations promote aggressive endometrial tumorigenesis

Fig 9

Model of independent and co-existing TP53 and ARID1A mutations in endometrial epithelia.

Summary of endometrial disease features and hypothesized molecular mechanisms in genetically engineered mouse models from this study.

doi: https://doi.org/10.1371/journal.pgen.1009986.g009