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Insulin secretion deficits in a Prader-Willi syndrome β-cell model are associated with a concerted downregulation of multiple endoplasmic reticulum chaperones
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Erik A. Koppes,
Roles Conceptualization, Data curation, Formal analysis, Investigation, Methodology, Visualization, Writing – original draft, Writing – review & editing
Affiliation Division of Genetic and Genomic Medicine, Department of Pediatrics, UPMC Children’s Hospital of Pittsburgh, Pittsburgh, Pennsylvania, United States of America
⨯ - Marie A. Johnson,
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James J. Moresco,
Roles Conceptualization, Data curation, Formal analysis, Investigation, Methodology, Writing – review & editing
Current address: Center for the Genetics of Host Defense, UT Southwestern Medical Center, Dallas, Texas, United States of America
Affiliation Department of Molecular Medicine and Neurobiology, The Scripps Research Institute, La Jolla, California, United States of America
⨯ - Patrizia Luppi,
- Dale W. Lewis,
- Donna B. Stolz,
- Jolene K. Diedrich,
- John R. Yates III,
- Ronald C. Wek,
- Simon C. Watkins,
- Susanne M. Gollin,
- Hyun Jung Park,
- Peter Drain,
- Robert D. Nicholls
https://orcid.org/0000-0002-4175-7777
Insulin secretion deficits in a Prader-Willi syndrome β-cell model are associated with a concerted downregulation of multiple endoplasmic reticulum chaperones
- Erik A. Koppes,
- Marie A. Johnson,
- James J. Moresco,
- Patrizia Luppi,
- Dale W. Lewis,
- Donna B. Stolz,
- Jolene K. Diedrich,
- John R. Yates III,
- Ronald C. Wek,
- Simon C. Watkins
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- Published: April 17, 2023
- https://doi.org/10.1371/journal.pgen.1010710