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Open Access
Peer-reviewed
Research Article
Inhibition of FLT1 ameliorates muscular dystrophy phenotype by increased vasculature in a mouse model of Duchenne muscular dystrophy
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Mayank Verma,
Roles Data curation, Formal analysis, Funding acquisition, Investigation, Methodology, Writing – original draft
Affiliations Medical Scientist Training Program, University of Minnesota Medical School, Minneapolis, MN, United States of America, Stem Cell Institute, University of Minnesota Medical School, Minneapolis, MN, United States of America, Paul & Sheila Wellstone Muscular Dystrophy Center, University of Minnesota Medical School, Minneapolis, MN, United States of America, Department of Neurology, University of Minnesota Medical School, Minneapolis, MN, United States of America
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Yuko Shimizu-Motohashi,
Roles Data curation, Formal analysis, Investigation
Affiliations Stem Cell Institute, University of Minnesota Medical School, Minneapolis, MN, United States of America, Paul & Sheila Wellstone Muscular Dystrophy Center, University of Minnesota Medical School, Minneapolis, MN, United States of America, Department of Neurology, University of Minnesota Medical School, Minneapolis, MN, United States of America
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Yoko Asakura,
Roles Data curation, Formal analysis, Investigation
Affiliations Stem Cell Institute, University of Minnesota Medical School, Minneapolis, MN, United States of America, Paul & Sheila Wellstone Muscular Dystrophy Center, University of Minnesota Medical School, Minneapolis, MN, United States of America, Department of Neurology, University of Minnesota Medical School, Minneapolis, MN, United States of America
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James P. Ennen,
Roles Data curation, Investigation
Affiliations Stem Cell Institute, University of Minnesota Medical School, Minneapolis, MN, United States of America, Paul & Sheila Wellstone Muscular Dystrophy Center, University of Minnesota Medical School, Minneapolis, MN, United States of America, Department of Neurology, University of Minnesota Medical School, Minneapolis, MN, United States of America
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Jennifer Bosco,
Roles Formal analysis, Investigation
Affiliation Shire Human Genetic Therapies, Inc., a member of the Takeda group of companies, Lexington, MA, United States of America
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Zhiwei Zhou,
Roles Formal analysis, Investigation
Affiliation Shire Human Genetic Therapies, Inc., a member of the Takeda group of companies, Lexington, MA, United States of America
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Guo-Hua Fong,
Roles Resources
Affiliation Center for Vascular Biology, University of Connecticut Health Center, University of Connecticut School of Medicine, Farmington, CT, United States of America
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Serene Josiah,
Roles Project administration, Resources
Affiliation Shire Human Genetic Therapies, Inc., a member of the Takeda group of companies, Lexington, MA, United States of America
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Dennis Keefe,
Roles Formal analysis, Project administration, Resources
Affiliation Shire Human Genetic Therapies, Inc., a member of the Takeda group of companies, Lexington, MA, United States of America
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Atsushi Asakura
Roles Conceptualization, Data curation, Formal analysis, Funding acquisition, Investigation, Project administration, Resources, Supervision, Validation, Writing – review & editing
* E-mail: asakura@umn.edu
Affiliations Stem Cell Institute, University of Minnesota Medical School, Minneapolis, MN, United States of America, Paul & Sheila Wellstone Muscular Dystrophy Center, University of Minnesota Medical School, Minneapolis, MN, United States of America, Department of Neurology, University of Minnesota Medical School, Minneapolis, MN, United States of America
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Inhibition of FLT1 ameliorates muscular dystrophy phenotype by increased vasculature in a mouse model of Duchenne muscular dystrophy
- Mayank Verma,
- Yuko Shimizu-Motohashi,
- Yoko Asakura,
- James P. Ennen,
- Jennifer Bosco,
- Zhiwei Zhou,
- Guo-Hua Fong,
- Serene Josiah,
- Dennis Keefe,
- Atsushi Asakura
- Published: December 26, 2019
- https://doi.org/10.1371/journal.pgen.1008468