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Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans
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Megan M. Senchuk ,
Contributed equally to this work with: Megan M. Senchuk, Dylan J. Dues
Roles Investigation, Supervision, Writing – review & editing
Affiliation Laboratory of Aging and Neurodegenerative Disease (LAND), Center for Neurodegenerative Science, Van Andel Research Institute, Grand Rapids, Michigan, United States of America
⨯ - Dylan J. Dues ,
- Claire E. Schaar,
- Benjamin K. Johnson,
- Zachary B. Madaj,
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Megan J. Bowman,
Roles Formal analysis, Writing – review & editing
Affiliation Bioinformatics and Biostatistics Core, Van Andel Research Institute, Grand Rapids, Michigan, United States of America
⨯ - Mary E. Winn,
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Jeremy M. Van Raamsdonk
Roles Conceptualization, Formal analysis, Funding acquisition, Investigation, Project administration, Supervision, Writing – original draft, Writing – review & editing
* E-mail: jeremy.vanraamsdonk@mcgill.ca
Affiliations Laboratory of Aging and Neurodegenerative Disease (LAND), Center for Neurodegenerative Science, Van Andel Research Institute, Grand Rapids, Michigan, United States of America, Department of Neurology and Neurosurgery, McGill University, Montreal, Quebec, Canada, Metabolic Disorders and Complications Program, and Brain Repair and Integrative Neuroscience Program, Research Institute of the McGill University Health Centre, Montreal, Quebec, Canada
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http://orcid.org/0000-0002-7744-2021
Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans
- Megan M. Senchuk,
- Dylan J. Dues,
- Claire E. Schaar,
- Benjamin K. Johnson,
- Zachary B. Madaj,
- Megan J. Bowman,
- Mary E. Winn,
- Jeremy M. Van Raamsdonk
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- Published: March 9, 2018
- https://doi.org/10.1371/journal.pgen.1007268