Truncation of Ube3a-ATS Unsilences Paternal Ube3a and Ameliorates Behavioral Defects in the Angelman Syndrome Mouse Model

Open Access

Peer-reviewed

Research Article

Linyan Meng,

Affiliation Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, United States of America
⨯
Richard Erwin Person,

Affiliation Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, United States of America
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Wei Huang,

Affiliation Department of Neuroscience, Baylor College of Medicine, Houston, Texas, United States of America
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Ping Jun Zhu,

Affiliation Department of Neuroscience, Baylor College of Medicine, Houston, Texas, United States of America
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Mauro Costa-Mattioli,

Affiliation Department of Neuroscience, Baylor College of Medicine, Houston, Texas, United States of America
⨯
Arthur L. Beaudet

* E-mail: abeaudet@bcm.edu

Affiliation Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, United States of America
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Mouse models
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Angelman syndrome
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Animal behavior
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Mammalian genomics
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Heterozygosity
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Genomic imprinting
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Polymerase chain reaction
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Prader-Willi syndrome
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