Contributions of Mamu-A*01 Status and TRIM5 Allele Expression, But Not CCL3L Copy Number Variation, to the Control of SIVmac251 Replication in Indian-Origin Rhesus Monkeys

Open Access

Peer-reviewed

Research Article

Contributions of Mamu-A01* Status and TRIM5 Allele Expression, But Not CCL3L Copy Number Variation, to the Control of SIVmac251 Replication in Indian-Origin Rhesus Monkeys

So-Yon Lim,

Affiliation Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, United States of America
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Tiffany Chan,

Affiliation Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, United States of America
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Rebecca S. Gelman,

Affiliation Dana Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts, United States of America
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James B. Whitney,

Affiliation Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, United States of America
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Kara L. O'Brien,

Affiliation Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, United States of America
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Dan H. Barouch,

Affiliation Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, United States of America
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David B. Goldstein,

Affiliation Center for Population Genomics and Pharmacogenetics, Duke Institute for Genome Sciences and Policy, Duke University, Durham, North Carolina, United States of America
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Barton F. Haynes,

Affiliation Duke Human Vaccine Institute, Duke University, Durham, North Carolina, United States of America
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Norman L. Letvin

* E-mail: nletvin@bidmc.harvard.edu

Affiliation Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, United States of America
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Rhesus monkeys
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SIV
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Monkeys
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HIV-1
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MHC class I genes
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Viral replication
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Viral load
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Copy number variation
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