Figures
In Fig 9C of [1], the Total H3, Cit H3, and β-actin panels are flipped along the horizontal axis. The corrected Fig 9 is provided with this notice, and the original blots are provided in [2].
B6 BMNs were left uninfected/UI or infected for 60 min with indicated strains at MOI 10 in the absence or presence of 5 mM glycine and analyzed for released IL-1β (A) or LDH (B). Data represent normalized values for 2.5x105 cells/well ± the standard deviation from three independent experiments. (C) Samples of total well contents were analyzed by immunoblotting for full length (FL-) or cleaved (N-) GSDMD, total histone 3 (H3), citrullinated histone 3 (Cit H3), and β-actin as a loading control. One representative blot of three independent experiments is shown. (A, B) Significant differences were determined by two-way ANOVA comparing p32_08 to UI or p32_08(ExsA-) or comparing p32_85 to p32_85 ExsA(A-) within groups or comparing between conditions as shown by brackets. ns, not significant; * P < 0.05; ** P < 0.01; *** P < 0.001; **** P < 0.0001.
References
- 1. Reuven AD, Katzenell S, Mwaura BW, Bliska JB. ExoS effector in Pseudomonas aeruginosa Hyperactive Type III secretion system mutant promotes enhanced Plasma Membrane Rupture in Neutrophils. PLoS Pathog. 2025;21(4):e1013021. pmid:40173191
- 2. Bliska J. Data associated with manuscript “Reuven, AD, Mwaura, BW, Bliska JB. ExoS effector in Pseudomonas aeruginosa hyperactive type III secretion system mutant promotes enhanced plasma membrane rupture in neutrophils.” [Dataset]. Zenodo; 2025 [Accessed June 2026].
Citation: Reuven AD, Katzenell S, Mwaura BW, Bliska JB (2026) Correction: ExoS effector in Pseudomonas aeruginosa Hyperactive Type III secretion system mutant promotes enhanced Plasma Membrane Rupture in Neutrophils. PLoS Pathog 22(6): e1014353. https://doi.org/10.1371/journal.ppat.1014353
Published: June 22, 2026
Copyright: © 2026 Reuven et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.