Figures
A natural killer cell contacting a P. falciparum infected erythrocyte.
Scanning electron microscopy image of a human natural killer (NK) cell (in blue) contacting a P. falciparum infected erythrocyte (red) in the presence of antibodies from individuals exposed to malaria. The initial contact shown here precedes the formation of tight immunological synapses that depend on specific receptor–ligand interactions between NK cells and infected erythrocytes. NK cells inflict damage to the erythrocyte plasma membrane and kill parasites that develop within intracellular vacuoles. Sekar et al 2023
Image Credit: David Dorward and Anita Mora (Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases)
Citation: (2023) PLoS Pathogens Issue Image | Vol. 19(11) December 2023. PLoS Pathog 19(11): ev19.i11. https://doi.org/10.1371/image.ppat.v19.i11
Published: December 1, 2023
Copyright: © 2023 . This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Scanning electron microscopy image of a human natural killer (NK) cell (in blue) contacting a P. falciparum infected erythrocyte (red) in the presence of antibodies from individuals exposed to malaria. The initial contact shown here precedes the formation of tight immunological synapses that depend on specific receptor–ligand interactions between NK cells and infected erythrocytes. NK cells inflict damage to the erythrocyte plasma membrane and kill parasites that develop within intracellular vacuoles. Sekar et al 2023
Image Credit: David Dorward and Anita Mora (Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases)