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Reversion mutations in phosphoprotein P of a codon-pair-deoptimized human respiratory syncytial virus confer increased transcription, immunogenicity, and genetic stability without loss of attenuation
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Jessica W. Chen,
Roles Data curation, Formal analysis, Investigation, Methodology, Visualization, Writing – original draft, Writing – review & editing
Current address: Ragon Institute of MGH, MIT, and Harvard, Cambridge, Massachusetts, United States of America.
Affiliation RNA Viruses Section, Laboratory of Infectious Diseases, NIAID, NIH, Bethesda, Maryland, United States of America
⨯ - Lijuan Yang,
- Celia Santos,
- Sergio A. Hassan,
- Peter L. Collins,
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Ursula J. Buchholz,
Roles Conceptualization, Data curation, Formal analysis, Methodology, Supervision, Visualization, Writing – review & editing
Affiliation RNA Viruses Section, Laboratory of Infectious Diseases, NIAID, NIH, Bethesda, Maryland, United States of America
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Cyril Le Nouën
Roles Conceptualization, Data curation, Formal analysis, Investigation, Methodology, Supervision, Visualization, Writing – original draft, Writing – review & editing
* E-mail: lenouenc@niaid.nih.gov
Affiliation RNA Viruses Section, Laboratory of Infectious Diseases, NIAID, NIH, Bethesda, Maryland, United States of America
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https://orcid.org/0000-0002-9095-1496
Reversion mutations in phosphoprotein P of a codon-pair-deoptimized human respiratory syncytial virus confer increased transcription, immunogenicity, and genetic stability without loss of attenuation
- Jessica W. Chen,
- Lijuan Yang,
- Celia Santos,
- Sergio A. Hassan,
- Peter L. Collins,
- Ursula J. Buchholz,
- Cyril Le Nouën
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- Published: December 29, 2021
- https://doi.org/10.1371/journal.ppat.1010191