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Open Access
Peer-reviewed
Research Article
Endocytosis of flavivirus NS1 is required for NS1-mediated endothelial hyperpermeability and is abolished by a single N-glycosylation site mutation
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Chunling Wang ,
Contributed equally to this work with: Chunling Wang, Henry Puerta-Guardo, Scott B. Biering
Roles Conceptualization, Formal analysis, Investigation, Methodology, Visualization, Writing – original draft, Writing – review & editing
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Henry Puerta-Guardo ,
Contributed equally to this work with: Chunling Wang, Henry Puerta-Guardo, Scott B. Biering
Roles Conceptualization, Formal analysis, Investigation, Methodology, Visualization, Writing – review & editing
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Scott B. Biering ,
Contributed equally to this work with: Chunling Wang, Henry Puerta-Guardo, Scott B. Biering
Roles Conceptualization, Formal analysis, Investigation, Methodology, Visualization, Writing – original draft, Writing – review & editing
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Dustin R. Glasner,
Roles Conceptualization, Formal analysis, Investigation, Methodology, Visualization, Writing – original draft, Writing – review & editing
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Edwina B. Tran,
Roles Investigation
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Mark Patana,
Roles Investigation
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Trent A. Gomberg,
Roles Investigation
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Carmel Malvar,
Roles Investigation
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Nicholas T. N. Lo,
Roles Investigation, Methodology
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Diego A. Espinosa,
Roles Formal analysis, Investigation, Methodology
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Eva Harris
Roles Conceptualization, Formal analysis, Funding acquisition, Methodology, Project administration, Resources, Supervision, Visualization, Writing – original draft, Writing – review & editing
* E-mail: eharris@berkeley.edu
Affiliation Division of Infectious Diseases and Vaccinology, School of Public Health, University of California, Berkeley, Berkeley, California, United States of America
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Endocytosis of flavivirus NS1 is required for NS1-mediated endothelial hyperpermeability and is abolished by a single N-glycosylation site mutation
- Chunling Wang,
- Henry Puerta-Guardo,
- Scott B. Biering,
- Dustin R. Glasner,
- Edwina B. Tran,
- Mark Patana,
- Trent A. Gomberg,
- Carmel Malvar,
- Nicholas T. N. Lo,
- Diego A. Espinosa
![PLOS](/resource/img/logo-plos.png)
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- Published: July 29, 2019
- https://doi.org/10.1371/journal.ppat.1007938