Epigenetic Repression of p16INK4A by Latent Epstein-Barr Virus Requires the Interaction of EBNA3A and EBNA3C with CtBP

Open Access

Peer-reviewed

Research Article

Epigenetic Repression of p16^INK4A by Latent Epstein-Barr Virus Requires the Interaction of EBNA3A and EBNA3C with CtBP

Lenka Skalska ,

Contributed equally to this work with: Lenka Skalska, Robert E. White

Affiliation Section of Virology, Division of Infectious Diseases, Faculty of Medicine, Imperial College London, London, United Kingdom
⨯
Robert E. White ,

Contributed equally to this work with: Lenka Skalska, Robert E. White

Affiliation Section of Virology, Division of Infectious Diseases, Faculty of Medicine, Imperial College London, London, United Kingdom
⨯
Melanie Franz,

Affiliation Section of Virology, Division of Infectious Diseases, Faculty of Medicine, Imperial College London, London, United Kingdom
⨯
Michaela Ruhmann,

Affiliation Section of Virology, Division of Infectious Diseases, Faculty of Medicine, Imperial College London, London, United Kingdom
⨯
Martin J. Allday

* E-mail: m.allday@imperial.ac.uk

Affiliation Section of Virology, Division of Infectious Diseases, Faculty of Medicine, Imperial College London, London, United Kingdom
⨯

B cells
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Chromatin
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DNA methylation
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Epigenetics
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DNA transcription
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Genetic loci
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Histones
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Fibroblasts
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