Figures
The flower of a tulip mutant with dysfunctional class B floral homeotic genes.
Petals and stamens are partially transformed into sepals and carpels, respectively. On the left, the graphical representations of gene regulatory networks help visualize how the obligate heterodimerization of class B floral homeotic genes may have arisen. One ancestral self-activating gene (top) is duplicated (middle), and subsequently, both transcription factors lose their homodimerization ability, leading to obligate heterodimerization (bottom) (see Lenser et al., doi:10.1371/journal.pcbi.1000264). The latter mechanism is found in Arabidopsis thaliana and other angiosperms.
Image Credit: Günter Theißen and Thorsten Lenser (University of Jena).
Citation: (2009) PLoS Computational Biology Issue Image | Vol. 5(1) January 2009. PLoS Comput Biol 5(1): ev05.i01. https://doi.org/10.1371/image.pcbi.v05.i01
Published: January 30, 2009
Copyright: © 2009 Lenser et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Petals and stamens are partially transformed into sepals and carpels, respectively. On the left, the graphical representations of gene regulatory networks help visualize how the obligate heterodimerization of class B floral homeotic genes may have arisen. One ancestral self-activating gene (top) is duplicated (middle), and subsequently, both transcription factors lose their homodimerization ability, leading to obligate heterodimerization (bottom) (see Lenser et al., doi:10.1371/journal.pcbi.1000264). The latter mechanism is found in Arabidopsis thaliana and other angiosperms.
Image Credit: Günter Theißen and Thorsten Lenser (University of Jena).