A Multi-Variant, Viral Dynamic Model of Genotype 1 HCV to Assess the in vivo Evolution of Protease-Inhibitor Resistant Variants
Figure 8
Objective values for alternative equations representing enhanced infected-cell clearance rates δ (Equations 5, 6, and 7).
Each of the models have the same number of parameters estimated for each subject. Models with δi that depend on blockage ε (Equations 5 and 6) have similar quality of fits. However, the model without dependency to ε (Equation 7) has inferior quality of fits.