Disruption of the ER-α36-EGFR/HER2 Positive Regulatory Loops Restores Tamoxifen Sensitivity in Tamoxifen Resistance Breast Cancer Cells

Open Access

Peer-reviewed

Research Article

Li Yin,

Affiliations Departments of Medical Microbiology and Immunology, Creighton University Medical School, Omaha, Nebraska, United States of America, Institute of Pathology and Southwest Cancer Center, Southwest Hospital, Third Military Medical University, Chongqing, China
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Xin-Tian Zhang,

Affiliation Departments of Medical Microbiology and Immunology, Creighton University Medical School, Omaha, Nebraska, United States of America
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Xiu-Wu Bian,

Affiliation Institute of Pathology and Southwest Cancer Center, Southwest Hospital, Third Military Medical University, Chongqing, China
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Yu-Ming Guo,

Affiliation Departments of Medical Microbiology and Immunology, Creighton University Medical School, Omaha, Nebraska, United States of America
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Zhao-Yi Wang

* E-mail: zywang@creighton.edu

Affiliation Departments of Medical Microbiology and Immunology, Creighton University Medical School, Omaha, Nebraska, United States of America
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Breast cancer
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Cancer treatment
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Cell disruption
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Kinase inhibitors
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Cell enumeration techniques
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Chemical disruption
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EGFR signaling
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Transfection
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