Sitagliptin Reduces Cardiac Apoptosis, Hypertrophy and Fibrosis Primarily by Insulin-Dependent Mechanisms in Experimental type-II Diabetes. Potential Roles of GLP-1 Isoforms

Open Access

Peer-reviewed

Research Article

Belén Picatoste,

Affiliation Instituto de Investigaciones Sanitarias-Fundación Jiménez Díaz, Madrid, Spain
⨯
Elisa Ramírez,

Affiliation Instituto de Investigaciones Sanitarias-Fundación Jiménez Díaz, Madrid, Spain
⨯
Alicia Caro-Vadillo,

Affiliation Veterinary School, Complutense University, Madrid, Spain
⨯
Cristian Iborra,

Affiliation Instituto de Investigaciones Sanitarias-Fundación Jiménez Díaz, Madrid, Spain
⨯
Jesús Egido,

Affiliation Instituto de Investigaciones Sanitarias-Fundación Jiménez Díaz, Madrid, Spain
⨯
José Tuñón ,

Contributed equally to this work with: José Tuñón, Óscar Lorenzo

Affiliation Instituto de Investigaciones Sanitarias-Fundación Jiménez Díaz, Madrid, Spain
⨯
Óscar Lorenzo

Contributed equally to this work with: José Tuñón, Óscar Lorenzo

* E-mail: olorenzo@fjd.es

Affiliation Instituto de Investigaciones Sanitarias-Fundación Jiménez Díaz, Madrid, Spain
⨯

Reader Comments (0)

Cardiomyocytes
Is the Subject Area "Cardiomyocytes" applicable to this article?

Thanks for your feedback.
Heart
Is the Subject Area "Heart" applicable to this article?

Thanks for your feedback.
Fibroblasts
Is the Subject Area "Fibroblasts" applicable to this article?

Thanks for your feedback.
Glucose
Is the Subject Area "Glucose" applicable to this article?

Thanks for your feedback.
Fibrosis
Is the Subject Area "Fibrosis" applicable to this article?

Thanks for your feedback.
Apoptosis
Is the Subject Area "Apoptosis" applicable to this article?

Thanks for your feedback.
Diabetes mellitus
Is the Subject Area "Diabetes mellitus" applicable to this article?

Thanks for your feedback.
Myocardium
Is the Subject Area "Myocardium" applicable to this article?

Thanks for your feedback.