Advertisement
Browse Subject Areas
?

Click through the PLOS taxonomy to find articles in your field.

For more information about PLOS Subject Areas, click here.

  • Loading metrics

Correction: Sitagliptin Reduces Cardiac Apoptosis, Hypertrophy and Fibrosis Primarily by Insulin-Dependent Mechanisms in Experimental type-II Diabetes. Potential Roles of GLP-1 Isoforms

  • The PLOS ONE Staff

Correction: Sitagliptin Reduces Cardiac Apoptosis, Hypertrophy and Fibrosis Primarily by Insulin-Dependent Mechanisms in Experimental type-II Diabetes. Potential Roles of GLP-1 Isoforms

  • The PLOS ONE Staff
PLOS
x

There was an error in affiliation number 1 for the first, second, fourth, fifth, sixth, seventh and last authors. The correct affiliation is: IIS-Fundación Jiménez Díaz, Autónoma University, Madrid.

In addition, an affiliation for the last author was incorrectly omitted from the byline. In addition to institution number 1, Óscar Lorenzo is also affiliated with the following institution: Spanish Biomedical Research Centre in Diabetes and Associated Metabolic Disorders (CIBERDEM).

Reference

  1. 1. Picatoste B, Ramírez E, Caro-Vadillo A, Iborra C, Egido J, et al. (2013) Sitagliptin Reduces Cardiac Apoptosis, Hypertrophy and Fibrosis Primarily by Insulin-Dependent Mechanisms in Experimental type-II Diabetes. Potential Roles of GLP-1 Isoforms. PLoS ONE 8(10): e78330