Table 1.
A summary of the demographic characteristics of 67 children investigated including their gender, age at baseline, and pre-treatment egg counts (average eggs per gram of feces determined by Kato-Katz) at each of the four annual treatment periods.
Table 2.
Temporal estimates of schistosome burdens of fifteen school aged children enrolled in a mass drug administration program in which they are treated annually.
Figure 1.
Mean number of schistosome eggs per gram of feces in patients for four years during annual mass praziquantel administration to school aged children.
A. 15 patients deemed “phenotypically susceptible” to schistosomiasis during MDA from which genetic samples were collected. Differences were due to a decrease in prevalence rather than a reduction in egg counts in infected individuals B. 52 randomly sampled children. Note the difference in scale of the Y-axis of both figures as egg burdens were much higher in the phenotypically susceptible group.
Figure 2.
Mean changes in genetic estimators of schistosome worm burdens collected from humans during four years of an annual mass treatment program.
Lines indicate means. A. number of full sibling families standardized according to sample size. B. effective number of breeders as estimated using the sibling assignment method.
Figure 3.
Mean changes in genetic diversity of schistosomes collected from humans before and after four years of an annual mass treatment program.
Lines indicate means. Data are corrected for sibling structure. A. allelic richness B. gene diversity.