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Figure 1.

Schematic representation of the recombinant VEEV TC-83/IRES constructs.

Alternative positions of the EMCV IRES are indicated. Arrows represent functional subgenomic promoters.

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Figure 2.

RNA synthesis and replication of VEEV TC-83 and TC-83/IRES constructs in BHK cells after electroporation.

(A) Analysis of 3[H]-labeled viral RNA production and (B) viral replication after transfection of 4 µg of in vitro-synthesized RNA into BHK cells. Dark dashed line indicates the limit of detection for the experiment.

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Figure 3.

Replication of recombinant TC-83/IRES viruses and parental TC-83 on Vero cells.

(A) Cells were infected with VEEV TC-83, VEEV/mutSG/IRES/1 and VEEV/IRES/C at an MOI of 0.1 then viral titers in collected supernatants over time were determined by plaque assay. Bars indicate standard deviation for duplicate infections. (B) Vero cells were fixed with 10% formaldehyde 48 h post-infection and stained with crystal violet to observed plaque size and morphology.

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Figure 4.

Survival in mice after infection with VEEV/IRES/C or VEEV TC-83 passaged in-vivo.

Six-day-old CD1 pups received a 5×104 PFU dose SC of viruses passaged 10 times in CD1 mice (mp10). Parent unpassaged VEEV TC-83 and VEEV/IRES/C were injected at the same dose as controls. Animals were monitored daily for survival for 14 days. No deaths occurred after day 11 post-infection. *** = P<0.0001.

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Figure 5.

Replication of recombinant TC-83/IRES viruses in mosquito cells.

(A) Five serial passages were performed on C6/36 cells at an initial MOI of 0.1 with VEEV/IRES/C, as well as VEEV TC-83 as a control. Supernatants were analyzed by plaque assay for detection of viral replication. Dark dashed line indicates the limit of detection for the experiment. (B) Supernatants were subjected to RT-PCR analysis to detect presence of viral RNA. Supernatant from non-infected cells (NI) was used as negative control.

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Table 1.

Virus replication in Ae. aegypti mosquitoes.

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Figure 6.

Virulence in mice after injection of VEEV TC-83 and IRES-based viruses.

Six-day-old CD1 mice received 5×104 PFU of indicated viruses SC, and were monitored 2 weeks for survival (A) and weight change (B). No deaths were recorded after day 14 post-infection.

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Figure 7.

Neurovirulence in 6-day-old mice following IC injection of VEEV TC-83 and IRES-based viruses.

Animals received 1×106 PFU of indicated viruses, and were monitored for survival (A) and weight change (B) for 2 weeks, with no deaths recorded after day 14 post-infection.

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Figure 8.

Protection against challenge following vaccination of infant mice.

Six-day-old mice were inoculated SC with 5×104 PFU of VEEV TC-83 or IRES-based viruses. Animals were challenged 6 weeks post-vaccination with 104 PFU SC of VEEV IC strain 3908 and monitored daily for survival (A) and weight change (B), with no deaths recorded after day 8 post-challenge. *** = P<0.0001.

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Table 2.

Seroconversion of infant CD1 mice.

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Table 2 Expand

Figure 9.

Survival following vaccination and challenge of adult mice.

Five-week-old CD1 mice were vaccinated with 105 PFU of VEEV TC-83 or IRES-based viruses. Challenge was performed 6 weeks post-vaccination by SC inoculation of 104 PFU of VEEV IC strain 3908, with daily monitoring of animals. No deaths occurred after day 11 post-challenge.

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Table 3.

Seroconversion of adult mice after vaccination.

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Table 4.

Viremia in vaccinated adult mice after challenge.

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