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Open Access

Correction

Correction: Cellular repressor of E1A-stimulated genes 1 enhances skeletal muscle performance through the stimulation of muscle differentiation and Akt-mTOR signaling pathway activation

  • Ayumi Goto,
  • Michihiro Hashimoto,
  • Sho Yokogawa,
  • Yuzu Naruse,
  • Hitoshi Yamashita

Fig 2D is a duplicate of Fig 2E. The authors have provided a corrected version of Fig 2 here.

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Fig 2. Muscle fiber-type composition and myosin heavy chain isoforms in wild-type (WT) and adipocyte P2-CREG1-transgenic (Tg) mice.

Muscle fiber-type composition was analyzed in the soleus and plantaris muscles of 5-month-old WT and Tg mice. (A-B) Immunostaining of myofibers in the soleus (A) and plantaris (B) muscles of WT and Tg mice. Type I (blue), type IIa (green), type IIx (black), type IIb (red), and lamin (white). Scale bar, 50 µm. (C-D) Distribution of muscle fiber types in the soleus (C) and plantaris (D) muscles. (E-F) Number of myofibers in the soleus (E) and plantaris (F) muscles of WT and Tg mice. (G-H) Relative levels of Myh7, Myh2, Myh1 and Myh4 mRNA in the soleus (G) and plantaris (H) muscles. Data were normalized to the 36b4 mRNA level. Data are presented as mean ± SEM; n = 4–6 per group. A student’s t-test was performed; *P < 0.05, versus WT.

https://doi.org/10.1371/journal.pone.0356625.g002

Reference

  1. 1. Goto A, Hashimoto M, Yokogawa S, Naruse Y, Yamashita H. Cellular repressor of E1A-stimulated genes 1 enhances skeletal muscle performance through the stimulation of muscle differentiation and Akt-mTOR signaling pathway activation. PLoS One. 2025;20(7):e0328485. pmid:40674409

Citation: Goto A, Hashimoto M, Yokogawa S, Naruse Y, Yamashita H (2026) Correction: Cellular repressor of E1A-stimulated genes 1 enhances skeletal muscle performance through the stimulation of muscle differentiation and Akt-mTOR signaling pathway activation. PLoS One 21(8): e0356625. https://doi.org/10.1371/journal.pone.0356625

Published: August 18, 2026

Copyright: © 2026 Goto et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.