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Correction

Correction: Hsa-mir-3163 and CCNB1 may be potential biomarkers and therapeutic targets for androgen receptor positive triple-negative breast cancer

  • The PLOS One Editors

There are errors in the caption for Fig 7. Please see the complete, correct Fig 7 caption here.

Fig 7 reports material from the Human Protein Atlas (proteinatlas.org), published under a Creative Commons Attribution-ShareAlike 4.0 International license [2]. No changes were made to the reused content. The images in Fig 7 are not offered under a CC BY license and are therefore excluded from this article’s [1] license.

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Fig 7. THPA website analysis of CCNB1 protein in BC. CCNB1 protein expression in BC specimens and non-cancerous breast tissues via THPA website analysis.

Three representative images of BC (images available from https://www.proteinatlas.org/ENSG00000134057-CCNB1/pathology/breast+cancer#img) and non-cancerous breast tissues were presented (images available from https://www.proteinatlas.org/ENSG00000134057-CCNB1/tissue/breast#img). Image Credit: Human Protein Atlas. N, normal; T, tumor; BC, breast cancer.

https://doi.org/10.1371/journal.pone.0352946.g007

References

  1. 1. Qiu P, Guo Q, Yao Q, Chen J, Lin J. Hsa-mir-3163 and CCNB1 may be potential biomarkers and therapeutic targets for androgen receptor positive triple-negative breast cancer. PLoS One. 2021;16(11):e0254283. pmid:34797837
  2. 2. Creative Commons. CC BY-SA 4.0 Attribution-ShareAlike 4.0 International [Internet] Available from: https://creativecommons.org/licenses/by-sa/4.0/ (Accessed June 2026)

Citation: The PLOS One Editors (2026) Correction: Hsa-mir-3163 and CCNB1 may be potential biomarkers and therapeutic targets for androgen receptor positive triple-negative breast cancer. PLoS One 21(7): e0352946. https://doi.org/10.1371/journal.pone.0352946

Published: July 6, 2026

Copyright: © 2026 The PLOS One Editors. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.