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Open Access

Correction

Correction: Inhibition of the Growth Factor MDK/Midkine by a Novel Small Molecule Compound to Treat Non-Small Cell Lung Cancer

  • Huifang Hao,
  • Yutaka Maeda,
  • Takuya Fukazawa,
  • Tomoki Yamatsuji,
  • Munenori Takaoka,
  • Xiao-Hong Bao,
  • Junji Matsuoka,
  • Tatsuo Okui,
  • Tsuyoshi Shimo,
  • Nagio Takigawa,
  • Yasuko Tomono,
  • Motowo Nakajima,
  • Iris M. Fink-Baldauf,
  • Sandra Nelson,
  • William Seibel,
  • Ruben Papoian,
  • Jeffrey A. Whitsett,
  •  [ ... ],
  • Yoshio Naomoto
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In Fig 5, the beta-actin is incorrect in panel B. Please see the correct Fig 5 here.

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Fig 5. iMDK inhibited the PI3K/AKT pathway and influenced the apoptosis pathway.

A. Dose-dependently, phosphorylation of PI3K and AKT and the expression of survivin and XIAP, anti-apoptotic factors, were decreased while the expression of BAD, a pro-apoptotic factor, was increased 48 hours after treatment with iMDK. Shown is immunoblot performed as described in Methods. B. Time-dependently, phosphorylation of PI3K and AKT and the expression of survivin and XIAP were decreased while the expression of BAD was increased by treatment with iMDK at a concentration of 50 nM. Immunoblot was performed as described in Methods.

https://doi.org/10.1371/journal.pone.0307052.g001

Reference

  1. 1. Hao H, Maeda Y, Fukazawa T, Yamatsuji T, Takaoka M, Bao X-H, et al. (2013) Inhibition of the Growth Factor MDK/Midkine by a Novel Small Molecule Compound to Treat Non-Small Cell Lung Cancer. PLoS ONE 8(8): e71093. pmid:23976985

Citation: Hao H, Maeda Y, Fukazawa T, Yamatsuji T, Takaoka M, Bao X-H, et al. (2024) Correction: Inhibition of the Growth Factor MDK/Midkine by a Novel Small Molecule Compound to Treat Non-Small Cell Lung Cancer. PLoS ONE 19(7): e0307052. https://doi.org/10.1371/journal.pone.0307052

Published: July 9, 2024

Copyright: © 2024 Hao et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.