Advertisement
Browse Subject Areas
?

Click through the PLOS taxonomy to find articles in your field.

For more information about PLOS Subject Areas, click here.

  • Loading metrics

Open Access

Correction

Correction: Role of the high-affinity leukotriene B4 receptor signaling in fibrosis after unilateral ureteral obstruction in mice

  • Mariko Kamata,
  • Hideki Amano,
  • Yoshiya Ito,
  • Tomoe Fujita,
  • Fumisato Otaka,
  • Kanako Hosono,
  • Kouju Kamata,
  • Yasuo Takeuchi,
  • Takehiko Yokomizo,
  • Takao Shimizu,
  • Masataka Majima

The Funding statement is incorrect. The correct Funding statement is as follows: This study was supported by Grants-in-Aid for Scientific Research, grant numbers: 17K16093 to Dr. Mariko Kamata; 15H05904, 18H02627 to Dr. Takehiko Yokomizo; 23116102 to Dr. Masataka Majima; 26462132 to Dr. Hideki Amano; and 26293055, 18H02605 to Dr. Masataka Majima.

Reference

  1. 1. Kamata M, Amano H, Ito Y, Fujita T, Otaka F, Hosono K, et al. (2019) Role of the high-affinity leukotriene B4 receptor signaling in fibrosis after unilateral ureteral obstruction in mice. PLoS ONE 14(2): e0202842. https://doi.org/10.1371/journal.pone.0202842 pmid:30818366

Citation: Kamata M, Amano H, Ito Y, Fujita T, Otaka F, Hosono K, et al. (2019) Correction: Role of the high-affinity leukotriene B4 receptor signaling in fibrosis after unilateral ureteral obstruction in mice. PLoS ONE 14(4): e0215625. https://doi.org/10.1371/journal.pone.0215625

Published: April 15, 2019

Copyright: © 2019 Kamata et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.