Figures
The first author’s name appears incorrectly in the article citation. The correct citation is: Wittenberg Dreazen A, Azar S, Klochendler A, Stolovich-Rain M, Avraham S, Birnbaum L, et al. (2016) Phosphorylated Ribosomal Protein S6 Is Required for Akt-Driven Hyperplasia and Malignant Transformation, but Not for Hypertrophy, Aneuploidy and Hyperfunction of Pancreatic β-Cells. PLoS ONE 11(2): e0149995. doi:10.1371/journal.pone.0149995.
Table 1 appears incorrectly in the published article. The last six rows are duplicates of the six above them. Please see the correct Table 1 and its caption below. The publisher apologizes for the error.
Whole cell extract from HEK293 cells infected with pS6[5S]-GFP, pS6[5A]-GFP, pS6[5D]-GFP or pEGFP-N1, was subjected to GFP pull-down, and the bound proteins were size fractionated by SDS-polyacrlamide gel electrophoresis. Mass spectrometric analysis of proteins in each lane was performed as described in “material and Methods” and proteins, selectively bound to pS6[5A]-GFP in two independent experiments, are presented (numbers separated by slash [/] represent results obtained in each of the two individual analyses).
Reference
- 1. Wittenberg AD, Azar S, Klochendler A, Stolovich-Rain M, Avraham S, Birnbaum L, et al. (2016) Phosphorylated Ribosomal Protein S6 Is Required for Akt-Driven Hyperplasia and Malignant Transformation, but Not for Hypertrophy, Aneuploidy and Hyperfunction of Pancreatic β-Cells. PLoS ONE 11(2): e0149995. pmid:26919188
Citation: The PLOS ONE Staff (2016) Correction: Phosphorylated Ribosomal Protein S6 Is Required for Akt-Driven Hyperplasia and Malignant Transformation, but Not for Hypertrophy, Aneuploidy and Hyperfunction of Pancreatic β-Cells. PLoS ONE 11(5): e0155281. https://doi.org/10.1371/journal.pone.0155281
Published: May 4, 2016
Copyright: © 2016 The PLOS ONE Staff. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.