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Correction: Avian Reovirus Protein p17 Functions as a Nucleoporin Tpr Suppressor Leading to Activation of p53, p21 and PTEN and Inactivation of PI3K/AKT/mTOR and ERK Signaling Pathways

  • Wei-Ru Huang,
  • Hung-Chuan Chiu,
  • Tsai-Ling Liao,
  • Kuo-Pin Chuang,
  • Wing-Ling Shih,
  • Hung-Jen Liu

The following information is missing from the Funding section: This project was supported by the Taichung Veterans General Hospital and National Chung Hsing University (TCVGH-NCHU1047602).

Two references are omitted from the first sentence of the “p17 Drives β-Arrestin-Mediated PTEN Translocation from the Cytoplasm to the Plasma Membrane via a Rock-1 Dependent Manner” subsection of the Results.

The sentence should read: It is well-known that the small GTPase, RhoA, was one of the known proteins to increase PTEN lipid phosphatase activity via its downstream effector Rho kinase (ROCK) [51, 52, Lima-Fernandes et al., 2011, Li et al., 2005].

The references are: Lima-Fernandes E, Enslen H, Camand E, Kotelevets L, Boularan C, Achour L, et al. (2011) Distinct functional outputs of PTEN signalling are controlled by dynamic association with β-arrestins. EMBO J. 30(13):2557–68.

Li Z, Dong X, Wang Z, Liu W, Deng N, Ding Y, et al. (2005) Regulation of PTEN by Rho small GTPases. Nat Cell Biol. 7(4):399–404.

Table 1 has been corrected for improved readability. Please see the corrected Table 1 here.

Reference

  1. 1. Huang W-R, Chiu H-C, Liao T-L, Chuang K-P, Shih W-L, Liu H-J (2015) Avian Reovirus Protein p17 Functions as a Nucleoporin Tpr Suppressor Leading to Activation of p53, p21 and PTEN and Inactivation of PI3K/AKT/mTOR and ERK Signaling Pathways. PLoS ONE 10(8): e0133699. pmid:26244501