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Open Access

Correction

Correction: The Tinkerbell (Tink) Mutation Identifies the Dual-Specificity MAPK Phosphatase INDOLE-3-BUTYRIC ACID-RESPONSE5 (IBR5) as a Novel Regulator of Organ Size in Arabidopsis

  • Kim L. Johnson,
  • Sascha Ramm,
  • Christian Kappel,
  • Sally Ward,
  • Ottoline Leyser,
  • Tomoaki Sakamoto,
  • Tetsuya Kurata,
  • Michael W. Bevan,
  • Michael Lenhard

The IAA concentration in Fig 3 is incorrect and should be listed as 100nM IAA. The authors have provided a corrected version here.

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Fig 3. Root phenotype of ibr5 alleles compared to wild-type.

a. On standard growth media (top panel) the ibr5-3 allele is indistinguishable from the wild-type (Col) whereas in media containing 100nm IAA, the ibr5-3 allele is insensitive to the inhibition of root growth seen in the wild-type (bottom panel). b. The tink/ibr5-6 allele shows reduced root growth compared to Ler on media with or without 100nm IAA and displays a slightly significant difference (p value ≤ 0.3) in root growth inhibition on 100nm IAA compared to the wild-type (c). Scale is 1cm. Values are shown as mean ± SEM where n = 20.

https://doi.org/10.1371/journal.pone.0136482.g001

Reference

  1. 1. Johnson KL, Ramm S, Kappel C, Ward S, Leyser O, Sakamoto T, et al. (2015) The Tinkerbell (Tink) Mutation Identifies the Dual-Specificity MAPK Phosphatase INDOLE-3-BUTYRIC ACID-RESPONSE5 (IBR5) as a Novel Regulator of Organ Size in Arabidopsis. PLoS ONE 10(7): e0131103. pmid:26147117

Citation: Johnson KL, Ramm S, Kappel C, Ward S, Leyser O, Sakamoto T, et al. (2015) Correction: The Tinkerbell (Tink) Mutation Identifies the Dual-Specificity MAPK Phosphatase INDOLE-3-BUTYRIC ACID-RESPONSE5 (IBR5) as a Novel Regulator of Organ Size in Arabidopsis. PLoS ONE 10(8): e0136482. https://doi.org/10.1371/journal.pone.0136482

Published: August 20, 2015

Copyright: © 2015 Johnson et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited