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Table 1.

Detailed parameters of each GWAS data in the structural equation modeling.

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Fig 1.

Flowchart detailing the study design and analytical pipeline.

The workflow encompasses the data preprocessing of single-input GWAS for four age-related eye diseases, multivariable Genomic Structural Equation Modeling (Genomic-SEM) to extract the latent ARED factor, and comprehensive post-processing analyses. SC: Senile cataract, Gla: Glaucoma; AMD: Age-related macular degeneration; DR: Diabetic retinopathy; GWAS: Genome-Wide Association Study; MAGMA: Multi-marker Analysis of GenoMic Annotation; TWAS: Transcriptome-Wide Association Study. (By Figdraw.).

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Fig 2.

Genomic risk loci identification and annotation for the latent ARED factor using FUMA.

A Manhattan plot of GWAS summary statistics (only SNPs with P-value ≤ 1 × 10−5 are kept) B Functional consequences of SNPs on genes.C Summary per genomic risk locus. D-F Risky genetic seat locations detected through FUMA. (rs1502593, rs2183836, rs3763764) GWAS: Genome-Wide Association Study; SNPs: single nucleotide polymorphisms.

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Fig 3.

The results of fine mapping and transcriptome prediction.

A,B Fine localization analysis identified strong associations at multiple genomic locations (mean.PP > 0.95). (SOX2-OT, SLC24A3) C Manhattan plot of 2 genes that exceeded the criteria for correction for multiple comparisons from the TWAS. D,E FOCUS fine positioning analysis results. (MMAB, SLC24A3) TWAS: Transcriptome-Wide Association Study.

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Fig 4.

Transcriptomic and gene-based association analyses.

A Miami plot of Z-scores from the TWAS. B Manhattan plot of the gene-based test as computed by MAGMA based on GWAS summary statistics. Genome wide significance (red dashed line in the plot) was defined at P = 0.05/17766 = 2.814 × 10−6; TWAS: Transcriptome-Wide Association Study; MAGMA: Multi-marker Analysis of GenoMic Annotation; GWAS: Genome-Wide Association Study.

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Fig 5.

Disease ontology enrichment analysis using MendelVar.

The bubble plot illustrates the significant enrichment of mapped genes in specific Mendelian disease categories. The x-axis represents the number of genes overlapping with the specific disease ontology, and the y-axis lists the enriched disease terms. The color gradient of the bubbles reflects the empirical P-value, while the size of the bubbles corresponds to the ratio of gene overlap.

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