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Fig 1.

Entire workflow for nsSNPs screening in the SCN9A gene using computational tools.

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Fig 1 Expand

Fig 2.

Pie chart distribution of Single nucleotide polymorphisms (SNPs) in SCN9A gene.

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Fig 3.

Prediction of functional consequences of nsSNPs by A) SIFT and B) PolyPhen.

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Table 1.

High risk non-synonymous SNPs identified by SIFT, Polyphen, Mutpred2 and SNAP2.

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Table 1 Expand

Table 2.

Cumulative prediction of possible deleterious nature of nsSNPs.

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Table 2 Expand

Table 3.

Prediction of disease causing SNPs by P-Mutant, Meta SNP, PhD SNP and SNP & GO.

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Table 3 Expand

Table 4.

List of nsSNP’s predicted by Mu-Pro, I mutant and ConSURF.

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Table 4 Expand

Fig 4.

Pie chart displaying the prevalence of deleterious missense mutations.

Evaluation of 15 In silico tools reveals the percentage and numerical quantity of deleterious nsSNPs.

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Fig 4 Expand

Fig 5.

Procheck-RAMACHANDRAN plot of the native SCN9A predicted model.

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Table 5.

Structural validation and comparison of SCN9A gene.

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Table 5 Expand

Table 6.

Top ranked binding affinities of 20 compounds with native and mutant proteins.

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Table 6 Expand

Fig 6.

Interaction of protein ligands with typical SCN9A and mutant D1778N, F1782V, L1802P, M939V, and V1311M.

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Fig 6 Expand

Table 7.

Interacting residues obtained from docking.

Wild protein, L1802P, F1782V, D1778N, V1311M and M936V variants with ligands including their binding residues and hydrophobic interactions.

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Table 7 Expand