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Table 1.

Plant material used for mapping and marker testing.

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Table 1 Expand

Fig 1.

Frequency distribution of disease severity.

Evaluation of resistance of the LP97-F2 population and control plants (A) and of Eagle-F1 and Eagle-F2 plants (B) to Pepper yellow leaf curl virus (PepYLCV) Indonesia strain performed 6 weeks after transfer of seedlings to the screen house. The disease severity index from 0 to 4 was used.

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Fig 1 Expand

Fig 2.

Genome-wide distribution of SNPs and bins across pepper genomes.

The outermost box with scale represents the 12 pepper chromosomes. The green histogram represents the density of raw SNPs; the blue histogram indicates the SNPs that are polymorphic between LP97 and ECW30R that were used for bin construction; the red histogram indicates the bins of adjacent SNPs that were used for linkage-map construction.

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Fig 2 Expand

Fig 3.

Collinearity between the genetic map and the physical map of pepper.

The outer circle represents the total number of chromosomes (chr = ID) on left side and linkage groups (chr = ID) on right side; markers located within linkage groups are linked to the parallel position on chromosomes by different colored lines in the inner circle.

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Fig 3 Expand

Table 2.

Summary of the bins and genetic linkage map for the LP97-F2 population.

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Table 2 Expand

Fig 4.

A bin based linkage map showing the locations of QTLs for resistance to PepYLCV.

The genetic distance is shown in centimorgans (cM). LP97-F2 plants were evaluated for resistance to PepYLCV Indonesia strain in a screen house.

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Fig 4 Expand

Table 3.

QTL analysis for resistance to PepYLCV Indonesia strain in the LP97-F2 population using composite interval mapping.

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Table 3 Expand

Table 4.

Candidate genes associated with PepYLCV resistance and their functions.

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Table 4 Expand

Fig 5.

Box plots of tightly linked bins to QTLs from chromosome P7 and P12 of LP97-F2.

A–C) LP97-F2 grouped based on the tightly linked bin to QTL peplcv-7 against PepYLCV Indonesia strain. D) LP97-F2 grouped based on the tightly linked bin to QTL peplcv-12 against PepYLCV Indonesia strain.

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Fig 5 Expand

Fig 6.

Validation of single markers.

A) Parental survey with marker Chr7-LCV-7 on P7. B) Parental survey with marker Chr12-LCV-12 on P12. C) Marker Chr7-LCV-7 on P7 validation using resistant hybrids. D) Marker Chr12-LCV-12 on P12 validation using resistant hybrids. E) Marker Chr7-LCV-7 on P7 validation using additional F2 segregating population Egale-F2. F) Marker Chr12-LCV-12 on P12 validation using additional F2 segregating population Eagle-F2. G) Single marker Chr7-LCV-7 on P7 genotype–phenotype association in Eagle-F2. H) Single marker Chr12-LCV-12 on P12 genotype–phenotype association in the Eagle-F2 population. (G & H) Green, blue and grey represents exact phenotype-genotype matched, when heterozygous counted as susceptible and unmatched, respectively.

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Fig 6 Expand

Table 5.

Marker sequence information used for QTL validation and genotyping.

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Table 5 Expand