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Table 1.

Demographics and clinical characteristics of the study population (n = 66 kidney transplant procedures performed in 65 patients).

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Table 2.

Immunosuppression and prophylaxis regimens (n = 66 kidney transplant procedures performed in 65 patients).

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Fig 1.

Temporal distribution of episodes of acute AMR in the study cohort.

AMR: antibody-mediated rejection.

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Fig 2.

Description of therapeutic approaches in 75 episodes of acute AMR according to individual agents or therapies used.

AMR: antibody-mediated rejection; ATG: antithymocyte globulin (different formulations [ATG-Fresenius® or Thymoglobulin®]); IA: immunoadsorption; IVIg: intravenous immunoglobulins; PPh: plasmapheresis.

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Table 3.

Clinical syndromes and causative agents of the episodes of infection occurring within the first 6 months after the diagnosis of acute AMR (n = 96).

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Fig 3.

Cumulative incidence of infection within the first 6 months after the diagnosis of acute AMR in the “per-patient” analysis.

(a) overall infection according to the use of plasmapheresis as anti-rejection therapy (log-rank test P-value = 0.002); (b) bacterial infection according to the use of IVIg as anti-rejection therapy (log-rank test P-value = 0.035). The last episode was taken as reference in patients with more than one rejection episode. AMR: antibody-mediated rejection; IVIg: intravenous immunoglobulins.

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Table 4.

Univariate and multivariate analyses of risk factors predicting the occurrence of overall infection within the first 6 months after the diagnosis of acute AMR in the “per-patient” analysis (i.e. the last episode was taken as reference in patients with more than one rejection episode).

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Table 5.

Univariate and multivariate analyses of risk factors predicting the occurrence of bacterial infection within the first 6 months after the diagnosis of acute AMR in the “per-patient” analysis (i.e. the last episode was taken as reference in patients with more than one rejection episode).

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Table 6.

Univariate and multivariate analyses of risk factors predicting the occurrence of opportunistic infection within the first 6 months after the diagnosis of acute AMR in the “per-episode” analysis (i.e. follow-up was censored at the time of diagnosis of the second or consecutive episodes in patients with more than one rejection episode).

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Fig 4.

Patient (a) and death-censored graft survival (b) within the first year after the diagnosis of acute AMR in the “per-patient” analysis. The last episode was taken as reference in patients with more than one rejection episode. AMR: antibody-mediated rejection.

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