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Table 1.

Patient characteristics and neuroimaging findings of pediatric patients attending ACSH (January 2016-August 2019).

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Table 2.

Characteristics of pediatric patients with cerebral palsy who underwent neuroimaging at ACSH between January 2016 and August 2019.

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Table 3.

Patterns of neuroimaging findings based on type of CP from January 2016 to August 2019.

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Table 4.

Distribution of Neuroimaging findings based on CP type from January 2016 to August 2019.

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Fig 1.

Types of cerebral palsy of patients who underwent neuroimaging at ACSH between January 2016 and August 2019.

Majority of the patients had spastic quadriplegia (33.3%) followed by spastic hemiplegia/hemiparesis (24.2%) and hypotonic CP (12.1%). 9.1% of the patients had spastic diplegia while patients with choreoathetoid and monoparetic CP constituted 6.1% and 3% each. 12.1% of the patients had unspecified forms.

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Fig 2.

Distribution of neuroimaging pathology among pediatric patients with cerebral palsy from January 2016 to August 2019.

Neuroimaging anomalies included pathologies of the white matter (18.2%), basal ganglia (15.2%), cortex and lobes (27.3%), corpus callosum (6.1%), lateral ventricle (12.1%), cysts (18.2%) and cerebellum (3%), respectively. Other findings were found in 45.5% of the patients.

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Fig 3.

MRI of an 11 month old patient with spastic diplegia.

MRI of an 11 month old male patient born at term with spastic diplegia (Case 31) showing deep right occipital horn posteriorly with temporal/frontal minimal increment in CSF spaces including cisterns (T2 FLAIR).

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Fig 4.

MRI of a 13 year old with right spastic hemiparesis.

MRI of a 13 year old male patient born at term with right spastic hemiparesis (Case 20) showing cortical and subcortical atrophy with increased subarachnoid space and adjacent T2 hyperintensity; most likely due to a perinatal stroke.

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Fig 5.

Case 30: MRI of a 3 year old patient with choreoathetoid CP.

MRI of a 3 year old female patient born at term presenting with choreoathetoid CP, showing bilateral frontal lobe volume loss compared with the temporal-parietal and occipital formations and Bilateral basal ganglia posterior hyperintensity on axial T2 FLAIR.

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Fig 6.

Case 19: MRI of a 3 year old male patient with unspecified form.

MRI images of a 3 year old male child delivered at term who did not cry immediately after birth. Axial T2 FLAIR MR images showing bilateral basal ganglia and thalamic hyperintensities.

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