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Table 1.

Demographic and clinical characteristics of enrolled patients.

Data are given as N (%) or median (1st - 3rd quartile).

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Fig 1.

Distribution of liver histology findings in our cohort.

Steatosis, lobular inflammation, ballooning of any grade, and fibrosis of any stage were present in 53%, 40%, 18% and 100% patients, respectively. Advanced degree of steatosis (≥2), lobular inflammation (≥2), ballooning (≥2) or fibrosis (≥3) was present in 13%, 8%, 2% and 18% patients, respectively.

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Fig 1 Expand

Table 2.

Associations of miRNAs with liver biopsy findings.

Top row shows fold changes and 95% confidence intervals. Bottom row shows p-values derived from 1-way ANOVA. Statistically significant results are printed in bold.

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Table 2 Expand

Table 3.

Steatosis.

Univariate analysis of the effect of clinical and laboratory findings on developing graft steatosis. Data are given as N (%) or median (1st - 3rd quartile). Significant results are printed in bold. Normal ranges of biochemical values are mentioned in Table 1. Non-steatosis group includes subjects without histologically proven graft steatosis (≤ 5% of hepatocytes); steatosis group comprises all patients with steatosis grade 1–3.

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Table 4.

Steatosis.

Multivariate logistic regression involving all significant variables from univariate analysis (including miR-33a). ALT underwent logarithmic transformation.

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Table 5.

Lobular inflammation.

Univariate analysis of the effect of clinical and laboratory findings on developing liver graft inflammation. Data are given as N (%) or median (1st - 3rd quartile). Significant results are printed in bold. Normal ranges of biochemical values are mentioned in Table 1. Non-lobular inflammation group includes subjects without histologically proven lobular inflammation; lobular inflammation group comprises all patients with lobular inflammation grade 1–3.

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Table 6.

Lobular inflammation.

Multivariate logistic regression involving all significant variables from univariate analysis (including miR-33a, miR-34a, miR-122).

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Fig 2.

ROC curve for liver graft steatosis.

ROC curve for steatosis shows that exclusion of miR-33a from the model decreased the accuracy of the regression model by 0.7%.

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Fig 2 Expand

Fig 3.

ROC curve for lobular inflammation.

ROC curve for liver graft inflammation shows that exclusion of miR-33a from the model decreased the accuracy of the regression model by 1%.

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