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Fig 1.

Survival prediction training and performance tasks.

Training-Task: Historical data with event times and censor-status along with covariates are used to train a model—top-to-bottom. Performance-Task: new patient covariates are input to the learned model to produce a prediction of survival time—bottom, left-to-right. (Included picture designed by Freepik.).

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Table 1.

Characteristics of METABRIC and KIPAN cohorts.

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Fig 2.

The dashed line is the Kaplan-Meier plot for this dataset.

Each of the other 5 curves is a patient-specific survival curve, for 5 different METABRIC patients, from a learned MTLR model. The curves show that there are very different prognoses for the different patients, even though they all are breast cancer patients from the same cohort: here, the patient with the orange survival curve (near the top) has a very good prognosis, especially compared to the patient with the blue survival curve.

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Fig 3.

Overview of learning process.

LSM = LearnSurvivalModel Uses Ψ ∈ {MTLR, Cox, RCox} for type of learner; ρ ∈ {dLDA, PCA} for the type of basis; XGE are the gene expression values, XCF are clinical features for a set of patients, “Lbl” is the set of their (survival prediction) labels, is the basis, of type ρ based on the instance XGE; W is the learned survival model, of type Ψ; and Ω is information about the preprocessing. Each (unrounded) box corresponds to data, whose dimensions appear around it. Each row is a patient, and each column, a feature. Each rounded box is a subroutine; here we show the input and output of each.

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Fig 4.

Histogram of the normalized Gene Expression values , from METABRIC, showing how we descretized them into essentially equal-width bins.

Note the heights are on a log-scale. The material under the histogram—involving x and y—compare two ways to compute the discretized Gene Expression Values (dGEV): The top Enc_A discretizes the GEVs into a single count-feature A(⋅), and the bottom Enc_B discretizes the GEVs into two count-features B(⋅) representing over-expression and under-expression, respectively.

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Fig 5.

The ComputeBasis[ρ=dLDA] process (shown top-to-bottom, on left) uses a set of high-dimensional gene expression vectors from many patients, to produce a set of basis vectors (corresponding the parameters of the LDA) .

The UseBasis[ρ=dLDA] process (shown left-to-right horizontally) uses the set of dLDA “basis vectors” to transform a gene expression vector from a novel patient, into a low-dimensional description . (Note ComputeBasis[ρ=dLDA] also uses other information, and Lbl, to determine the number K of basis vectors; to simplify the figure, we did not show this.).

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Fig 6.

Overview of performance process, USM = UseSurvivalModel.

xGE and xCF are the gene expression values and clinical feature values, for a single patient; see also terms ρ, Ψ, Ω, , W from Fig 3.

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Fig 7.

Test CI: METABRIC (left) and KIPAN (right).

Note higher values are better. The labels on the x-axis correspond to Table 2.

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Table 2.

Concordance results of various models from METABRIC (over 395 test instances) and KIPAN (over 176 test instances).

+ = used these features and − = did not use these features As PAM50 is specific to breast cancer, it is not applicable to the kidney (KIPAN) data. The first row, with the ID “A(*)”, is the baseline.

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Table 3.

D-calibration results from METABRIC, KIPAN, and BRCA, on the held-out test data.

p-values greater than 0.05 suggest the model is good (“D-calibrated”). Note that Cox models fail D-calibration test for all feature combinations, for BRCA dataset.

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Fig 8.

Simplified flow diagram describing the overall prediction process, including both the learning task and the performance task.

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Fig 9.

Kaplan–Meier survival function from METABRIC (training) data.

(We only use the quartiles for pedagogic purposes.).

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