Table 1.
Demographics, exposure, immune cell phenotypes and T cell subsets of a male Bangladeshi Study Cohort.
Fig 1.
Covariate-adjusted associations between UAs/Cr and cell surface markers.
Individual data points represent observations; solid lines represent estimated mean outcome vs. UAs/Cr adjusted for other variables in the model. The outcomes (A-B) were Th (CD4+) cells and monocytes (CD14+) and outcomes (C-D) were a subset of monocytes (CD14+CD16+) and NKT cells in logarithmic scales.
Fig 2.
Covariate-adjusted associations between PAHs and cell surface markers.
Individual data points represent observations; solid lines represent estimated mean outcome vs. PAH-DNA adducts adjusted for other variables in the model. The outcomes (A-D) were T (CD3+), Th (CD4+), monocytes (CD14+), and Tmem (CD3+CD45+) cells in logarithmic scale.
Table 2.
Association between urinary arsenic concentration per creatinine (UAs/Cr) and immune cell phenotypes (n = 179).
Table 3.
Association between PAH-DNA adducts and immune cell phenotypes (n = 179).
Fig 3.
Covariate-adjusted association between UAs/Cr and Th17 cells.
Individual data points represent observations; solid line represent estimated mean outcome Th171/3 vs. UAs/Cr adjusted for other variables in the model.
Fig 4.
Covariate-adjusted association between PAH-DNA adducts and Th, Th1, Th2 and Th17 cells.
Individual data points represent observations; solid lines represent estimated mean outcome vs. PAH-DNA adducts adjusted for other variables in the model. The outcome variables (A-D) were Th, Th11/2, log(Th2), and Th171/3.
Table 4.
Association between urinary arsenic concentration per creatinine (UAs/Cr) and T cell subsets (n = 180).
Table 5.
Association between PAH-DNA adducts and T cell subsets (n = 180).