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Table 1.

Clinicobiological features of the 11 EBV+ DLBCL patients analyzed by whole-exome sequencing (WES).

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Table 2.

Patient characteristics at diagnosis of EBV+ DLBCL (11 cases).

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Fig 1.

Mutation spectrum of Epstein-Barr virus (EBV)-positive diffuse large B-cell lymphoma shown by whole-exome sequencing.

Frequency of substitutions in each sample for the six possible classes of mutation. The most common substitution was the transition C>T/G>A, followed by T>C/A>G.

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Fig 2.

Distribution of mutations in different genome regions of Epstein-Barr virus (EBV)-positive diffuse large B-cell lymphoma detected by whole-exome sequencing.

(A) The percentages of mutations in different regions of EBV+ DLBCL cases. The percentage of mutations in exonic regions ranged from 3.49%–22.68% among these 11 cases. (B) We identified 3326 protein-coding genes with somatic mutations affecting exonic regions, with a range from 200 to 410 per case.

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Table 3.

Comparison of the numbers of mutated genes in exonic regions, and ratio to total mutations detected by WES in 11 EBV+ DLBCL cases according to age and pathological subtype.

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Fig 3.

An overview of the data analysis strategy for single-nucleotide variations (SNVs) and indels shown by whole-exome sequencing.

On average, we identified 220 (range, 126–358) somatically acquired point mutations per case. Of these, an average of 168 (range, 105–252) were non-synonymous and 81 (range, 52–149) were synonymous. Thus, the ratio of non-synonymous to synonymous was 2.08 (0.95–2.67) mutations per Mb.

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Fig 4.

Signatures of mutational processes and intratumor clonal heterogeneity of EBV+ DLBCL cases.

The DeconstructSigs method was applied, which uncovered four mutational signatures (Fig 4A), including signatures 3, 5, 12, and 30. Among these signatures, signature 3 was the main type. MATH scores were used to evaluate intratumor clonal heterogeneity (Fig 4B), revealing allele frequency distribution values of between 47 and 86 for the tumors.

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Table 4.

Relationship between clinical variables and MATH scores and OS in 11 EBV+ DLBCL cases.

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Fig 5.

The 30 most commonly mutated genes from selected variants in EBV+ DLBCL identified by whole-exome sequencing.

Based on the total number of all mutation loci within one somatic mutated gene in a detected region (exonic region, intronic region, intergenic region, UTR, and other regions), the top 5 most frequently mutated genes were MUC16, PRSS3, MUC19, MUC3A, and RLIM in decreasing order.

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