Table 1.
Baseline characteristics of patient population.
Table 2.
CMR characteristics.
Fig 1.
Anterior and inferior QTc derivation over time.
Anterior (panel A), inferior (panel B) QTc derivations and ΔQTc-AI-MA (panel C) at different time points expressed as mean and standard error. PCI: percutaneous coronary intervention; ΔQTc-AI-MA: max anterior QTc—min inferior QTc.
Fig 2.
ROC curve for ΔQT AI MA at day six versus MSI <60%.
ΔQTc AI MA: delta QT corrected antero-inferior max; MSI = myocardial salvage index.
Table 3.
Multivariate linear regression analysis for log-transformed LGE (gr).
Table 4.
Multivariate linear regression for log-transformed LGE (%).
Table 5.
Multivariate logistic regression for MSI <60%.
Fig 3.
Panel 1. A: ECG at admission. B: ECG at 6th day. C, E: T2-weighted images of basal and mid-short axis views, respectively. The increased myocardial signal intensity (arrows) indicates increased water content, hence tissue edema in the anterior, antero-septal and infero-septal walls. D, F: late gadolinium enhancement (LGE) images (basal and mid-short axis views respectively). Necrosis (arrows) and microvascular obstruction (arrowhead) are shown. The edematous myocardial content was 62gr, corresponding to 56% of total left ventricular mass, and LGE was 59gr, corresponding to 54% of total left ventricular mass. The myocardial salvage index (MSI) was 0.05. ΔQTc-AI-MA (max anterior QTc—min inferior QTc) was 200msec. Panel 2. A: ECG at admission. B: ECG at 6th day. C, E: T2-weighted images of basal and mid-short axis views, respectively. The increased myocardial signal intensity (arrows) indicates tissue edema in the anterior and antero-septal walls. D, F: LGE images (basal and mid-short axis views, respectively). The edematous myocardial content was 30gr, corresponding to 25% of total left ventricular mass. No LGE was evident. The MSI was 1. No ΔQTc-AI-MA (max anterior QTc—min inferior QTc) was present.