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Table 1.

Plasmids and strains used in this study.

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Fig 1.

Construction of the expression vector for production of the H2 antigen fused to the Lp_1261 lipoprotein anchor.

SalI and HindIII restriction sites enabled easy gene exchange. The gene fragment encoding the Hirep2-DC antigen (darker gray) was introduced into a plasmid harboring the lipoprotein anchor derived from L. plantarum protein Lp_1261 (light gray). The complete gene construct was translationally fused to the inducible promoter that is indicated by the thin arrow.

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Table 2.

Experimental groups and immunization protocol.

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Fig 2.

Detection of anchor-fused H2 antigen (A) and cell growth of L. plantarum producing the H2 antigen (B). Panel A shows Western blot analysis of a cell-free protein extract from Lp_H2 (lane 2) and a molecular mass standard (1). The predicted molecular mass of the Lp_1261H2-DC protein is 37 kDa. Panel B shows the growth rate of Lp_H2 and Lp_Ev, used as a control. The OD600 was measured at the induction point (black bars) and 3 h after induction (gray bars) for both strains. The data are presented as means of triplicates ± SEM.

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Fig 3.

Presence of the H2 antigen at the L. plantarum surface detected by flow cytometry (A) and fluorescence microscopy (B). Cells were probed with an anti-H2 antibody. Panel A shows flow cytometry analysis for Lp_H2 (black histogram) and Lp_Ev used as a negative control (gray histogram). Panel B shows indirect immunofluorescence microscopy of the indicated strains. The data presented are from one representative experiment. Each experiment was performed three independent times, giving similar results.

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Fig 4.

Estimation of the total amount of antigen by semi-quantitative Western blotting.

Serial dilutions of bacterial cell lysate from 1 x 109 CFU of induced Lp_H2 and dilutions of purified Hirep1 with known concentration were subjected to Western blotting and proteins were detected using the anti-H2 antibody (which also binds to Hirep1 protein). Panel A shows a typical Western blot: wells 1–5, standard protein (20 ng, 10 ng, 7.5 ng, 5 ng and 2.5 ng, respectively); wells 6–9, lysate of Lp_H2, diluted 20-fold, 30-fold, 40-fold and 50-fold, respectively. Signal intensities of different dilutions of the standard protein were used to make a standard curve that is presented in panel B. The marked points on the standard curve correspond with the amount of standard protein used: (1) - 20 ng, (2) - 10 ng, (3)– 7.5 ng, (4) - 5 ng, (5) - 2.5 ng.

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Fig 5.

Immune responses induced by the booster Lp_H2 vaccine.

Panel A presents the cellular response measured by H2-specific IFN-γ production in splenocytes. Cells were purified from individual mice and stimulated with H2 for 72 h. Produced IFN-γ was measured in harvested supernatants, by ELISA. Each point represents an individual mouse and the overall results per group are presented as a mean ± SEM (n = 8 for naive, H2 and H2/Lp_H2 groups, n = 7 for Lp_H2). Statistical significance was determined using one-way ANOVA with Tukey's post hoc test and is indicated as follows: **, p < 0.01. Panels B—E show antibody responses: H2-specific IgG in plasma (B) and H2-specific IgA in plasma (C), vaginal washes (D) and lung fluids (E). The samples from individual mice were serially diluted and added to H2-coated plates and specific IgG and IgA levels were measured by ELISA. The individual points represent the average OD450 values per group ± SEM (n = 8 for naive, H2 and H2/Lp_H2 groups, n = 7 for Lp_H2) at each dilution. Panels F and G show H2-specific IgA in 10-fold diluted plasma samples (F) and 5-fold diluted vaginal samples (G). Each point represents an individual mouse and the overall results per group are presented as a mean ± SEM (n = 8 for naive, H2 and H2/Lp_H2 groups, n = 7 for Lp_H2). Statistical significance was determined using one-way ANOVA with post hoc Dunnett's test and is shown as follows: *, p < 0.05; **, p < 0.01.

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