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Table 1.

Patient Collective: m = male, f = female, Age (at the time of operation), Duration since diagnosis (number of years since disease diagnosis).

Score of the motor part of the Unified Parkinson’s Disease Rating Scale (UPDRS) without medication: UPDRS Off, score of the motor part of the UPDRS with medication: UPDRS On. The UPDRS test was performed prior to surgery.

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Table 1 Expand

Fig 1.

Example of long-latency somatosensory evoked potential (LL-SSEP) Complex, Patient 4, right STN, monopolar reference, contralateral median nerve stimulation (MNS).

Black: Contact 0, red: Contact 1, blue: Contact 2, green: Contact 3.

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Fig 1 Expand

Fig 2.

Zoom-in view of Fig 1 emphasizing the short-latency somatosensory evoked potential (SL-SSEP) component N20: Patient 4, right STN, monopolar reference, contralateral median nerve stimulation (MNS).

Black: Contact 0, red: Contact 1, blue: Contact 2, green: Contact 3.

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Fig 2 Expand

Table 2.

Statistical comparison of mean latencies between the N18/20 in the present study and data reported in the literature revealed no significant difference (significance level of 0.05).

N denotes the number of nuclei with occurrence of the N18/20, Latency (ms): Mean ± standard deviation (SD) (range), Amplitude (μV): Mean ± SD (range), NA “not available”. This finding provides support for the used methodology, namely forehead rather than linked earlobes/mastoids for monopolar reference and filtering methods (Notch and 40 Hz high cut-off), in eliciting somatosensory evoked potentials (SSEPs).

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Table 2 Expand

Table 3.

Long-latency somatosensory evoked potential (LL-SSEP) components corresponding to monopolar data: N denotes the number of nuclei with a referred wave, Latency (ms): Mean ± SD (range), Amplitude (μV): Mean ± SD (range).

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Table 3 Expand

Fig 3.

Example of LL-SSEPs recorded with bipolar reference.

Left: Phase reversal of the P140 and the N200, Patient 2, left STN, ipsilateral MNS. Right: Maximum amplitude of the P80 and N140, contact (1 vs. 2), Patient 4, right STN, contralateral MNS.

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Fig 3 Expand

Table 4.

LL-SSEP components corresponding to bipolar data: N denotes the number of nuclei with a referred wave, Latency (ms): Mean ± SD (range), Amplitude (μV): Mean ± SD (range), “Total Phase reversals” denotes the total number of reversals across bipolar contacts for each LL-SSEP component and stimulation side, “Max 1–2” denotes the number of nuclei with maximum amplitude at bipolar contact (1 vs. 2).

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Table 4 Expand

Fig 4.

Example of LL-SSEPs recorded with bipolar reference.

Left: Phase reversal of the P80 at contact (0 vs. 1), Patient 9, right STN, ipsilateral MNS. Right: Phase reversal of the N100 at contact (2 vs. 3), Patient 2, left STN, contralateral MNS.

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Fig 4 Expand

Table 5.

Number of phase reversals for each bipolar contact, LL-SSEP component and stimulation side (ipsi- and contralateral).

No significant difference was found in the number of phase reversals between bipolar contacts according to the Wilcoxon signed-rank test (p = 0.157 for ((0 vs. 1) vs. (1 vs. 2)), p = 0.395 for ((1 vs. 2) vs. (2 vs. 3)), p = 1.0 for ((0 vs. 1) vs. (2 vs. 3))).

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Table 5 Expand