Table 1.
Demographics, clinical characteristics, and outcomes of PCP in renal transplant recipients vs. HIV-infected patients.
Table 2.
Location and pattern frequency of PCP manifestations in renal transplant recipients and HIV-positive patients.
Fig 1.
A) 45-year-old HIV-positive male patient with PCP with typical diffuse ground glass opacities in both lungs. B) 58-year-old male patient 5 years after kidney transplantation who developed PCP with multifocal patchy ground glass opacities and reticulation.
Fig 2.
HIV-positive individual (49-year-old, male), CDC stage C3 with PCP.
Faint, peripheral ground glass opacities with diffuse distribution in the upper and lower lobes in both lungs. This finding was significantly more frequent in HIV-positive individuals. Subpleural sparing is also present.
Fig 3.
55-year-old HIV positive male patient with PCP presenting with ground glass nodules in a diffuse distribution, predominantly in the right lower lobe (circles in image A and B).
This finding was significantly more prevalent in the HIV-positive study population. Patchy opacifications are also evident in the left lower lobe.
Fig 4.
70-year old, male RTR presenting with PCP.
Bilateral, patchy consolidations with areas of ground glass attenuation. Septal thickening in the outer portions of both lungs is present. Also, bilateral, peribronchial cuffing can be seen. Note the absence of hilar lymphadenopathy.