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Fig 1.

Proportion of recipient baits colonized by Rhizoctonia solani upon saprotrophic spread at (A) 10 mm, (B) 12.5 mm and (C) 25 mm after treatment with Monceren® L (pink lines) or tap water (blue lines).

Different letters indicate significant differences in the proportion of colonized baits (P-value < 0.05, Wald test).

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Fig 1 Expand

Table 1.

Values of the parameters of the model for saprotrophic spread and estimated distributions.

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Table 1 Expand

Fig 2.

Histogram describing the posterior distributions of the parameters of the saprotrophic spread model.

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Fig 2 Expand

Fig 3.

Saprozone dynamics for saprotrophic spread of Rhizoctonia solani, describing the change in the probability colonization for a bait placed at a given distance from a mycelium disc and after a given time of exposure.

Circles indicate data observed in the placement experiments.

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Fig 3 Expand

Fig 4.

Effect of fungicide treatment on pathogen infectivity.

Bars show the mean cumulated incidence at harvest (i.e. 30 days after sowing); dark and light grey refer to the type of symptom: damping-off and tuber necrosis, respectively. The analysis of deviance using a Wald chi-square test shows no significant differences between treatments.

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Fig 4 Expand

Fig 5.

Proportion of recipient baits colonized by Rhizoctonia solani upon pathogenic spread at (A) 25 mm, (B) 50 mm and (C) 75 mm after treatment with Monceren® L (pink lines) or tap water (blue lines).

Different letters indicate significant differences in the proportion of colonized baits (P-value < 0.05, Wald test).

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Fig 5 Expand

Fig 6.

Histogram describing the estimated distribution of the parameters of the pathogenic spread model.

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Fig 6 Expand

Fig 7.

Saprozone dynamics for pathogenic spread of Rhizoctonia solani, describing the change in the probability of colonization for bait placed at a given distance from a mycelium disc and after a given time of exposure.

Circles indicate data observed in the placement experiments. Data were fitted to the model.

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Fig 7 Expand

Table 2.

Values of the parameters of the model for pathogenic spread and estimated distributions.

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Table 2 Expand