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Fig 1.

CONSORT Flow Diagram.

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Fig 2.

mDA neurons derived from hESCs were treated with N-acetyl cysteine at 10 μM for 3 days before challenge with rotenone (15nM and 30 nM) for 24 hours.

TH+ neurons were quantified after cells were fixed and stained. Data was normalized as percentage of surviving mDA neurons compared to untreated control (100%). Treatment groups were compared with rotenone only groups using unpaired t-test. * p<0.05, ** p<0.01.

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Fig 2 Expand

Table 1.

Demographics for the study subjects per group.

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Table 2.

Mean pre- and post-Tx dopamine transporter binding measures.

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Table 2 Expand

Table 3.

Mean pre-to-post-Tx differences in dopamine transporter binding.

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Table 3 Expand

Fig 3.

Pre and post NAC DaTscans (left and right respectively) from one particularly responsive patient (A) shows a substantial increase in dopamine transporter binding in the basal ganglia (arrows). We also present (B) the paired t test results overlaid onto a standard MRI template using SPM8 software showing significantly greater binding (p<0.05) post NAC in the basal ganglia.

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Table 4.

Mean pre- and post-Tx UPDRS Total Score.

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Table 4 Expand

Table 5.

Significant predictors of UPDRS Total Score.

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Table 5 Expand