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Table 1.

In vivo experimental protocol.

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Table 1 Expand

Table 2.

Renal functional parameters of control and experimental groups.

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Table 2 Expand

Table 3.

Hemodynamic parameters of control and experimental groups.

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Table 3 Expand

Fig 1.

In vivo inhibition of OCTs with D-22 and its effects on Na+, K+-ATPase activity in the presence of dopamine (DA) and ANP plus DA.

*p<0.001 vs Control/P+T; **p<0.05 vs DA; ***p<0.05 vs ANP; #p<0.05 vs ANP plus DA; $p<0.05 vs D-22.

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Fig 1 Expand

Fig 2.

Effects of exogenous dopamine, ANP and D-22 on dopamine urinary levels.

*p<0.05 vs Control; **p<0.05 vs DA; #p<0.05 vs ANP plus DA.

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Fig 3.

A, B and C. Western blot analysis of OCT-1 (A), OCT-2 (B) and OCT-3 (C) in renal cortex: Effects of MAO and COMT inhibition by pargyline and tolcapone (P+T) and ANP infusion on OCT-1 (A), OCT-2 (B) and OCT-3 (C) protein expression in membrane preparations from renal cortex.

Histograms illustrate the values of protein expression of OCTs in each group, normalized to GAPDH expression.

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Fig 3 Expand

Fig 4.

Western blot analysis of D1R in renal cortex: Effects of MAO and COMT inhibition by pargyline and tolcapone (P+T) and ANP infusion on D1R expression in membrane preparations from renal cortex.

Histograms illustrate the values of protein expression of D1R in each group, normalized to GAPDH expression.

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Fig 4 Expand

Fig 5.

Effects of ANP, D-22, anantin and KT 5823 on 3H-dopamine uptake.

*p<0.05 vs Control; ***p<0.01 vs ANP; ##p<0.05 vs ANP.

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Fig 5 Expand

Fig 6.

Schematic representation of the mechanism by which ANP could enhance dopamine tubular transport in proximal tubule cells, by stimulation of organic cationic transporters.

A: under basal conditions, exogenous dopamine is carried by OCTs located at the basolateral membrane and uptaken from circulation and interstitium by the proximal tubular cells. B: Once inside the tubular cell, dopamine can reach the tubular lumen through the OCTNs located at the apical membrane. Atrial natriuretic peptide inhibits Na+, K+-ATPase activity through stimulation of NPR-A receptors, cGMP and PKG. C: ANP enhances dopamine tubular transport by OCTs, stimulating its specific activity, and increasing dopamine concentration at the luminal side. The simultaneous inhibition of Na+, K+-ATPase activity by ANP and dopamine promotes a greater natriuretic response. OCTs: organic cationic transporters. OCTNs: carnitine/organic cationic transporters. Black circles: dopamine; gray triangles: ANP. Full arrows: stimulation; dot arrows: inhibition; gray arrow with?: hypothetical mechanism.

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Fig 6 Expand