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Fig 1.

Chemical structures of the ATP analogs used in this study.

The structures of ATP, 7-deazaATP, 2'-deoxyATP and AMPPCP with key atom positions indicated. Boxes denote the region—nucleobase, sugar, triphosphate—containing the modification for that analog.

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Fig 1 Expand

Table 1.

Data collection and refinement statistics.

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Fig 2.

GRP78ATPase domain structure and canonical ATP/ADP binding site.

(A). Overview of the GRP78ATPase subdomain structure: Ia (cyan), Ib (yellow), IIa (purple), and IIb (green). (B). Close-up of ATP binding site indicates key interacting residues. (C). Overlay of the ATP (cyan) and ADP-Pi-Mg++ (green) binding sites highlights the difference between the ATP γ-phosphate and Pi. (D). Fourier difference maps (Fo-Fc) of ATP and ADP-Pi-Mg++ contoured to 2.5 σ. Dashed lines indicate potential hydrogen bonds (green) and metal coordination (black). Non-bonding spheres indicate water (blue) and magnesium (green). For clarity, the oxygen atoms of ADP are shown in magenta.

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Fig 2 Expand

Table 2.

GRP78 ATPase nucleotide affinities (Kd) determined by SPR.

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Table 2 Expand

Fig 3.

Binding site of AMPPCP.

(A). The binding site of AMPPCP-Mg++-bound (yellow) GRP78ATPase illustrates the key residues interacting with the phosphate groups. Asp224, which interacts with the other Mg++-coordinated waters, has been removed for clarity. Dashed lines indicate potential hydrogen bonds (green) and metal coordination (black). Non-bonding spheres indicate water (blue) and magnesium (green; from AMPPCP-Mg++). (B). Fourier difference map (Fo-Fc) of AMPPCP-Mg++ contoured to 3.0 σ; the arrow denotes the carbon bridging the β- and γ-phosphates. (C). Superposition of α-, β- and γ-phosphate groups in ATP (gray) and AMPPCP-Mg++ (orange and red). Residues correspond to the AMPPCP-Mg++-bound structure at the active site of GRP78ATPase. (D). Superposition of α-, β- and γ-phosphate/Pi groups in ADP-Pi (gray) and AMPPCP-Mg++ (orange and red) at the active site of GRP78ATPase. Residues correspond to the AMPPCP-Mg++-bound structure.

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Fig 4.

Binding site of 7-deazaATP.

(A) 7-deazaATP bound to GRP78ATPase illustrates that the 7-position of the adenine ring faces an opening (dash wedge) bordered by Ser300 and Arg367. (B). Overlay of ATP ligand (cyan) and 7-deazaATP-bound GRP78ATPase demonstrates the minimal effect of a 7-position modification. (C). Fourier difference map (Fo-Fc) of 7-deazaATP contoured to 3.0 σ. The arrow denotes the carbon at the 7-position of the adenine ring.

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Fig 5.

Binding site of 2'-deoxyATP.

(A). Surface representation of GRP78ATPase shows the outward binding mode of 2'-deoxyATP (magenta) and the inward binding mode of ATP (cyan). (B). Close-up of the 2'-deoxyATP binding site with superimposed ATP (cyan) shows key residues in grey. (C). Alternate view of 2'-deoxyATP binding site highlights interactions with Arg60, Arg367 and Asn389. (D). Fourier difference map (Fo-Fc) of 2'-deoxyATP contoured to 3.0 σ. Green dashed lines indicate potential hydrogen bonds.

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