Fig 1.
Model of megalin function in renal uptake and activation of 25-(OH) Vitamin D3. [11]
Fig 2.
Course of the study.
Table 1.
Patients characteristics at baseline.
CM: contrast media, VDBP: vitamin D binding protein, KIM-1: kidney injury molecule 1, uCr: urinary creatinine, CIN: contrast induced nephropathy, GFR: glomerulary filtration rate estimated with the MDRD formula, ME: mean, M: mean, SD: standard deviation,*—p<0.05 is statistically significant.
Table 2.
Time to death and causes of death during the follow up.
Table 3.
Urinary concentration of VDBP and KIM-1 24 hrs after CM injection comparison in patients with and without complications.
VDBP: vitamin D binding protein, KIM-1: kidney injury molecule 1, CIN: contrast induced nephropathy, MARE: major adverse renal event, GFR: glomerulary filtration rate estimated with the MDRD formula, N: number of patients, M: mean, SD: standard deviation, p: significance according to MANOVA, p<0.05 –is statistically significant.
Table 4.
VDBP/uCr, KIM-1/uCr 24 hrs after CM injection and baseline creatinine comparison in patients with and without complications.
VDBP: vitamin D binding protein, KIM-1: kidney injury molecule 1, uCr: urinary creatinine, CIN: contrast induced nephropathy, MARE: major adverse renal event, GFR: glomerulary filtration rate estimated with the MDRD formula, N: number of patients, M: mean, SD: standard deviation, p: significance according to MANOVA, p<0.05 –is statistically significant.
Table 5.
Logistic regression analyses MARE independent variables.
VDBP: vitamin D binding protein, KIM-1: kidney injury molecule 1, p<0.05 –is statistically significant.