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Fig 1.

Representative schematic structure of ManLAM, Insert in the Blue box show residues adapted as strategic surrogates for LAM.

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Fig 1 Expand

Fig 2.

Protocol for derivatization of D-Arabinose in urinary LAM and the corresponding mass fragmentation pattern.

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Fig 2 Expand

Fig 3.

NMR spectra of anomeric protons and GC/MS peak ratio and different stereoisomeric forms of 2,3,5-trifluoroacetyl-1-(R-2-octyl)-arabinosyl glycosides.

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Fig 3 Expand

Fig 4.

Differences in peak patterns and retention times in GC/MS chromatogram for D- and L- arabinose.

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Fig 4 Expand

Fig 5.

D-Arabinose estimation of Non-Endemic-Urine (NEU).

1) Internal standard, 2) D-arabinose in NEU before purification; 3) D-arabinose not detected in NEU after purification.

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Fig 5 Expand

Table 1.

FIND urine sample purification and D-arabinose analysis.

IS = Internal Standard; HIV = Human Immunodeficiency Virus; Pneu = Pneumonia; Atyp = Atypical TB,

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Table 1 Expand

Fig 6.

Protocol for TBSA detection by GC/MS.

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Fig 6 Expand

Fig 7.

GC/MS chromatogram for TBSA detection in TBSSMC+ and TBSSMC- urine samples.

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Fig 7 Expand

Table 2.

TBSA Based LAM Estimation of 29 Urine Samples (TBSSMC -, D-Arabinose/LAM positive).

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Table 2 Expand

Fig 8.

Summary of results for the GC/MS based urinary LAM detection on clinical samples.

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Fig 8 Expand