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Table 1.

Characteristics of all recruited patients.

Shown are means of the criteria ± SD.

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Table 2.

Data of selected study subjects (5 per group aseptic (NS) and SIRS).

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Table 2 Expand

Fig 1.

SIRS parameters of selected study patients.

(A) Body temperature, (B) heart rate, (C) breathing rate, (D) PaCO2 and (E) leukocyte counts are presented as means ± SEM per study group. Dotted lines indicate threshold values to pathological levels, shaded areas between dotted lines indicate the physiological ranges. * p < 0.05 Kruskal-Wallis with Dunn’s multiple comparison post-hoc test for selected data pairs.

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Fig 2.

Normalized changes in IL1β and IL10 plasma cytokine levels have potential predictive value for early recognition of SIRS.

Shown are fold changes ± SEM in plasma concentrations of (A) IL1β and (B) IL10 per patient, normalized to the day of admission, over the collected time points. * p < 0.05; ** p < 0.01; *** p < 0.001 Kruskal-Wallis with Dunn’s multiple comparison post-hoc test for selected data pairs. $ p < 0.05 Mann Whitney U-test.

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Fig 3.

Normalized plasma levels of endocan and cfDNA rise significantly slower whereas soluble TREM-1 plasma levels are upregulated at early time points in SIRS patients.

(A) Fold changes in endocan (ESM-1) ± SEM plasma levels of individual patients normalized to the corresponding plasma levels at admission. The fold increase is significantly lower in SIRS group than in control group ** p < 0.01 Kruskal-Wallis with Dunn’s multiple comparison post-hoc test for selected data pairs; $$ p < 0.01 Mann Whitney U-test. (B) Individual plasma levels of cfDNA ± SEM (expressed as fold changes over admission value) tend to increase during surgery but the rise is lower in SIRS than in the control group. * p < 0.05 Kruskal Wallis with Dunn’s multiple comparison post hoc test for selected pairs. (C) Soluble human triggering receptor expressed on myeloid cells-1 (sTREM-1) ± SEM plasma levels are significantly increased in SIRS patients at the end of surgery and day 2 after surgery compared to the control group. * p < 0.05; ** p < 0.01 Kruskal-Wallis with Dunn’s multiple comparison post hoc test for selected data pairs. A trend to an increased expression at the end of surgery within the SIRS group is also seen. p = 0.0556 Mann Whitney U-test.

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Fig 4.

Monocyte activation test (MAT) revealed reduced responsiveness in SIRS patients.

Cryopreserved blood of each patient at individual time points was stimulated with reference endotoxin (0.5 EU/ml) corresponding to 0.04 EU/ml in the actual incubation for 8 h at 37°C 5% CO2. (A) Individual stimulation responses (shown as IL1β concentrations ± SEM) of diluted cryopreserved patient blood to an LPS stimulus show a tendency towards decreased responses (p = 0.1508) at the end of surgery but increased responses one day after surgery (p = 0.0952) in the SIRS group; Mann Whitney U-test. (B) When the responses to the LPS stimulus are normalized to the responses on the admission day, there is a significant decrease at the end of surgery obvious in the SIRS but not in the control group. ** p < 0.01 Kruskal-Wallis with Dunn’s multiple comparison post hoc test for selected data pairs.

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