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Table 1.

Bacterial Strains and Plasmids.

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Fig 1.

Y. pestis crp-deficient Mutant 26°C Growth Kinetics.

(A) Growth curves of CO92, CO92Δcrp, and plasmid complemented CO92Δcrp: pBluescript/crp when grown in BCS medium supplemented with 0.2% glucose or 0.2% K-gluconate. Growth of the CO92 crp-deficient mutant when cultured in medium supplemented with 0.2% K-gluconate was significantly impaired relative to all other bacterial strain and media conditions (Tukey’s HSD P < 0.05). (B) Growth curves of KIM6+ and KIM6+Δcrp when cultured in BCS medium supplemented with either 0.2% glucose, 0.2% glycerol, or a combination of both 0.2% glucose and 0.2% glycerol. Growth of the KIM6+ crp-deficient mutant when cultured in medium solely supplemented with 0.2% glycerol was significantly impaired relative to all other bacterial strain and media conditions (Tukey’s HSD P < 0.05). (C) Growth kinetics of the CO92 and KIM6+ crp-deficient mutants and respective isogenic controls when grown in HIB medium at 26°C.

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Fig 2.

Y. pestis csrA-deficient Mutant 26°C Growth Kinetics.

(A) Growth of CO92, CO92ΔcsrA, and the chromosomal restoration mutant CO92ΔcsrA csrA’ in BCS medium supplemented with either 0.2% glucose or 0.2% K-gluconate during incubation at 26°C. (B) Growth kinetics of the CO92 and KIM6+ csrA-deficient mutants and respective controls when cultured in HIB medium at 26°C. No significant alteration in growth kinetics as determined by repeated measures ANOVA was calculated among the csrA-deficient mutants and the respective controls, regardless of media type or available carbon source.

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Fig 2 Expand

Fig 3.

Carbon Catabolite Regulation of Y. pestis Biofilm Production.

Crystal violet biofilm quantification assay of KIM6+ and CO92 when cultured in BCS medium supplemented with primary (glucose) and/or alternate (glycerol or K-gluconate) carbon sources. Error bars reflect standard deviation from the mean derived from two independent experiments, each consisting of 6 technical replicates. * P-value <0.005 determined by Tukey’s HSD post-hoc analysis.

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Fig 3 Expand

Fig 4.

CRP Enables Robust Y. pestis Biofilm Production.

Relative biofilm production of CO92, CO92Δcrp, and plasmid complemented CO92Δcrp: pBluescript/crp when cultured in HIB medium or BCS medium supplemented with either 0.2% K-gluconate or 0.2% glucose. (A) Following 24 hours post-inoculation. (B) After 72 hours post-inoculation. Error bars reflect standard deviation from the mean derived from two independent experiments, each consisting of 6 technical replicates. * P-value <0.005 determined by Tukey’s HSD post-hoc analysis.

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Fig 4 Expand

Fig 5.

Y. pestis Biofilm Production is not Influenced by Exogenous cAMP.

(A) Relative biofilm production of KIM6+ and KIM6+Δcrp when grown in BCS medium supplemented with 0.2% glucose containing either 3 mM cAMP prepared in K-phosphate buffer or an equal volume of plain K-phosphate buffer. (B) Relative biofilm production of CO92 and CO92Δcrp when grown in BCS medium supplemented with 0.2% glucose containing either 3 mM cAMP prepared in K-phosphate buffer or an equal volume of plain K-phosphate buffer. Identical source inoculum per strain was utilized for each media type. Error bars reflect standard deviation from the mean derived from two independent experiments, each consisting of 6 technical replicates. No statistically significant change in crystal violet absorption as determined by one-way ANOVA was calculated for both the crp-deficient mutants and the respective parental controls when cultured in media containing 3mM cAMP or an equal volume of K-phosphate buffer.

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Fig 5 Expand

Fig 6.

Semi-quantitative RT-PCR of the Y. pestis Hms System.

Depiction of semi-quantitative RT-PCR fold changes in expression of the Y. pestis Hms biofilm formation/regulation system following normalization to the gyrB gene. “OM” reflects the bacterial outer membrane; whereas, “IM” indicates the inner membrane. Factors highlighted in green represent characterized pro-biofilm factors. Factors colored in red have been shown to impair biofilm production. I. Fold changes in mRNA abundance amongst CO92 grown in BCS medium supplemented with 0.2% glucose to CO92 cultured in BCS supplemented with 0.2% K-gluconate. II. Fold changes in mRNA abundance of CO92 to CO92Δcrp when grown in HIB medium.

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Fig 6 Expand

Fig 7.

CsrA is a Positive Regulator of Y. pestis Biofilm Production.

Relative biofilm production of the csrA-deficient mutants after 24 hours inoculation of BCS medium supplemented with 0.2% K-gluconate, HIB medium, or BCS medium supplemented with 0.2% glucose. (A) CO92, CO92ΔcsrA, and the chromosomal restoration mutant CO92ΔcsrA csrA’. (B) KIM6+, KIM6+ΔcsrA, and the chromosomal restoration mutant KIM6+ΔcsrA csrA’. Results reflect the average of two independently derived csrA-deficient mutants (CO92 clones 5a and 3b, and KIM6+ clones 2:14 and 4:12). Two biological replicates were assessed per mutant, each consisting of 6 technical replicates. Error bars reflect standard deviation from the mean among biological replicates. * P-value < 0.005 as determined by Tukey’s HSD post-hoc analysis. (C) Impaired Congo red assimilation of the Y. pestis csrA-deficient mutants. Phenotypic evaluation of Congo red binding of the CO92 and KIM6+ csrA-deficient mutants, chromosomal csrA restoration mutants, parental isogenic controls, and pigmentation locus negative (biofilm-deficient) mutants after 48 hours post-inoculation of Congo red plates supplemented with 0.2% K-gluconate.

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Fig 7 Expand

Table 2.

Relative Crystal Violet Absorption of the CO92 Glycogen-deficient Mutants.

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Fig 8.

CsrA does not alter Y. pestis hmsH, hmsP, and hmsT Transcript Levels.

Relative mRNA abundance of hmsH, hmsP, and hmsT among the csrA-deficient mutants and the respective parental strains calculated via the ΔΔCT method in reference to the gyrB gene. Dashed lines reflect two-fold thresholds for alterations in relative transcript levels. No significant change in relative mRNA abundance among the deletion mutants and the respective controls were determined by one-way ANOVA comparing the average of 2 independent cDNA samples per strain, each consisting of 3 technical replicates per target.

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Fig 9.

Deletion of hmsP Enables Excessive Biofilm Production in the csrA-deficient Mutant.

Relative biofilm production of CO92, CO92ΔcsrA, CO92ΔhmsP, and CO92ΔcsrAΔhmsP following 24 hours post-inoculation in HIB medium. Error bars reflect standard deviation from the mean of 2 independent experiments, each consisting of 6 technical replicates. * P-value < 0.005 as determined by Tukey’s HSD post-hoc analysis.

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Fig 9 Expand