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Table 1.

Primer list.

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Table 2.

Mechanical properties for aligned PLGA nanofiber scaffolds.

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Fig 1.

The alignment of hiPSC-CMs.

Cells seeded on A) Flat surface plate and B) Aligned nanofiber characterized by cell anisotropic index (CAI). The hiPSC-CMs were stained with phalloidin dye (Red) to determine the alignment of seeded cells. CAI was significantly increased in the aligned nanofiber group.

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Fig 2.

Analysis of gap junctions in hiPSC-CMs by confocal microscopy in cells seeded on flat plate and aligned nanofiber.

The gap junctions in hiPSC-CMs was evaluated by CX-43 immunostaining of human cardiomyocytes seeded on flat surface versus aligned-nanofiber coated coverslips. A) Flat plate, CX-43 (Red), SAA (Green) and DAPI (Blue); B) Aligned nanofiber, CX-43 (Red), SAA (Green) and DAPI (Blue); C) Quantification of gap junctions (% CX-43 positive area fraction per cell) in hiPSC-CMs seeded on flat plate and aligned nanofiber. There were no significant differences between the two groups. All values expressed as mean ± SD (n = 4/group).

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Fig 3.

Confocal Imaging of mitochondria.

Mito-tracker red staining shows alignment of Human iPSC-Cardiomyocytes seeded on A) Flat surface vs B) Aligned-nanofiber coated coverslips (32x & 200x). C) TEM imaging showing comparison of mitochondrial morphology and arrangement of hiPSC-CMs seeded on flat plate versus aligned nanofiber groups.

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Fig 4.

SEM imaging of Human iPSC-Cardiomyocytes.

Cells seeded on A) Flat surface vs B) Aligned-nanofiber coated coverslips (600x & 1200x) show different cell morphology.

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Fig 5.

TEM imaging of Human iPSC-Cardiomyocytes.

Cells seeded on Flat surface A) nucleus, B) mitochondria, C) myofibrils into sarcomere. Cells seeded on aligned nanofiber D) nucleus, E) mitochondria and F) myofibrils, (n = 4; 4500x, 22500x & 34000x) demonstrated myofibrils well-organized into sarcomeres with clear aligned Z-discs.

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Fig 6.

Intracellular calcium cycling in hiPSC-CMs.

A) Line-scan images and temporal profiles of Fluo-4 fluorescence of hiPSC-CMs seeded on flat plate or on aligned nanofiber-coated coverslips. Ca+2 transients were evoked by electrical field stimulation at 0.5 Hz. B) Summary graphs showing average data for Ca+2 transient amplitude, time to peak and exponential decay constant (τ) of Ca+2 transients recorded in cells from flat plate (Flat, n = 10) and aligned nanofiber (Nano, n = 14) groups, respectively. *P<0.05 vs standard flat plate group. HiPSC-CMs cultured on aligned nanofibers have faster calcium cycling rate and higher contraction frequency than cells maintained on a flat surface.

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Fig 7.

Electrical properties of hiPSC-CMs.

A) Representative action potential (AP) traces recorded during 1 Hz stimulation in patch-clamped single human iPS-CM seeded on flat bottom or on aligned nanofiber-coated coverslips. Dotted lines indicate 0 mV level. B) Summary graphs showing average data for AP amplitude and AP duration at 50 (APD50) and 90 (APD90) % repolarization levels, respectively. Data was recorded in cells from flat plate (Flat, n = 5) and aligned nanofiber (Nano, n = 4) groups, respectively. Resting potential was -74± 2 mV in flat bottom, and -74± 1 mV in aligned nanofiber groups. C) Representative action potential (AP) traces recorded in syncytium of human iPS-CM seeded on flat bottom or on aligned nanofiber-coated coverslips. APs were evoked by electrical field stimulation at 1 Hz and were recorded using voltage-sensitive dye di-4-AN(F)EPPTEA. D) Summary graphs showing average data for AP amplitude and AP duration. Data was obtained both flat plate (Flat, n = 6) and aligned nanofiber (Nano, n = 6) preparations, respectively.

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Fig 8.

Assessment of cardiac genes by qRT-PCR.

mRNA levels of three cardiac markers connexin-43 (GJA1), a-actinin (ACTN2), and troponin-I (TNNC1). A) hiPSC-CMs had approximately two fold greater levels of GJA1 mRNA compared to human heart tissue (human 0.78 vs hiPS-CM on flat plates 2.69, n = 6, p = 0.001) B) No significant differences in a-actinin mRNA were found between groups C) hiPSC-CMs of both groups had approximately 1/3 the amount of troponin-I mRNA compared to non-failing human heart tissue (human 1.06 vs hiPSC-CMs on flat plates 0.38, n-6, p = 0.01).

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