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Figure 1.

Cytotoxicity of geniposide, borneol, muscone and their combinations determined by MTT test.

(A) Geniposide has no cell cytotoxicity in the concentration range of 0–400 µg·mL−1. (B) Borneol has no cell cytotoxicity in the concentration range of 0–300 µg·mL−1. (C) Muscone has no cell cytotoxicity in the concentration range of 0–70 µg·mL−1. (D) GB group (geniposide:borneol, 0.9∶1, w/w) has no cell cytotoxicity in the concentration range of 0–200 µg·mL−1 calculated by borneol. (E) GM group (geniposide:muscone, 6∶1, w/w) has no cell cytotoxicity in the concentration range of 0–40 µg·mL−1 calculated by muscone. (F) GBM group (geniposide:borneol:muscone, 0.9∶1∶0.15, w/w/w) has no cell cytotoxicity in the concentration range of 0–150 µg·mL−1 calculated by borneol. Date are expressed as mean ± SD (n = 5). * P<0.05 compared with the control group.

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Figure 1 Expand

Table 1.

Various concentrations of geniposide permeability across the HNEC monolayer.

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Table 1 Expand

Figure 2.

Effects of borneol (Bo) and muscone (Mu) on geniposide (Ge) transport across the HNEC monolayer.

(A) Geniposide (50 µg·mL−1) combined with borneol A to B transport. (B) Geniposide (50 µg·mL−1) combined with borneol B to A transport. (C) Geniposide (50 µg·mL−1) combined with muscone A to B transport. (D) Geniposide (50 µg·mL−1) combined with muscone B to A transport. (E) Geniposide (50 µg·mL−1) combined with borneol and muscone A to B transport. (F) Geniposide (50 µg·mL−1) combined with borneol and muscone B to A transport. Date are expressed as mean ± SD (n = 3).

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Figure 2 Expand

Table 2.

Effects of borneol (Bo) and muscone (Mu) on geniposide (Ge) transport in the HNEC monolayer.

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Table 2 Expand

Figure 3.

Actin distribution in HNEC monolayer treated with borneol (Bo) and muscone (Mu).

(A) 50 µg·mL−1 geniposide (control). (B) (C) and (D) 50 µg·mL−1 geniposide combined with 27.8, 55.6 and 111.2 µg·mL−1 borneol, respectively. (E) (F) and (G) 50 µg·mL−1 geniposide combined with 4.17, 8.34 and 16.68 µg·mL−1 muscone, respectively. (H) (I) and (J) 50 µg·mL−1 geniposide combined with borneol and muscone (Bo 27.8 and Mu 4.17 µg·mL−1, Bo 55.6 and Mu 8.34 µg·mL−1, Bo 111.2 and Mu 16.68 µg·mL−1, respectively). Scale bar: 20 µm.

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Figure 3 Expand

Figure 4.

TEER of HNEC monolayer before permeation, after permeation, and after 24 h.

Date are expressed as mean ± SD (n = 3). * P<0.05, TEER of after permeation compared with before permeation, ** P<0.05, TEER of after 24 h compared with after permeation.

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Figure 4 Expand

Figure 5.

Fluorescence images of membrane phospholipids of HNECs by tomoscan.

(A) Before photobleaching, the fluorescence on membrane phospholipids of HNEC was green and well-distributed. (B) After photobleaching, the fluorescence disappeared. (C) Due to membrane fluidity, the fluorescence probe from other areas can move to the photobleaching position leading to the recovery of fluorescence.

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Figure 5 Expand

Table 3.

Fluorescence recovery rate after treatment with borneol (Bo) and muscone (Mu).

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Table 3 Expand