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Figure 1.

Representative immunohistochemistry results of stromal Cav-1 expression in pancreatic tissue.

Paraffin sections were immunostained as described in the methods section. (N) Normal tissue strongly positive for Cav-1 expression; (P) Paracancer tissue moderate for Cav-1 expression; (C) Tumor tissue with negative Cav-1 expression (×400).

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Figure 2.

The mRNA expression of stromal Cav-1 in laser-captured samples by RT-PCR and Real-time PCR.

A: Schematic diagram representing the LCM technique. B: mRNA expression of stromal Cav-1 in laser-captured samples as determined by RT-PCR. C: Quantification of mRNA by real-time quantitative PCR. Data from at least three independent experiments with duplicate determinations are expressed as means ± SEM. (N) Normal tissue; (P) Paracancer tissue; (C) Tumor tissue. *P<0.05 was considered statistically significant.

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Figure 3.

Immunodetection of Cav-1 protien in CAFs, PAFs, and NFs.

A: Cell morphological characteristic of CAFs, PAFs, and NFs (×100). Extracted CAFs from pancreatic cancer and PAFs and NFs from paracancerous tissue and non-cancerous partial pancreatectomy specimens. B: Different vimentin expression levels in stroma CAFs, PAFs, and NFs were assessed by immunohistochemistry (×200). C: Cav-1 protein expression in CAFs and NFs stained with FITC labeling-IgG antibody and analyzed by confocal microscopy. Cav-1 fluorescence signal in CAF is lower than in PAFs and NFs (×200). (CAFs) Cancer associated fibroblasts; (PAFs) Paracancerous associated fibroblasts; (NFs) Normal associated fibroblasts.

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Table 1.

Association between stromal Cav-1 expression and clinicopathologic factors in pancreatic cancers.

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Figure 4.

Representative photomicrograph of HER-2/neu gene amplification in pancreatic cancer.

(N) Normal tissue negative for HER-2/neu gene amplification; (P) Paracancer tissue moderate for HER-2/neu gene amplification; (C) Tumor tissue positive for HER-2/neu gene amplification (×1000).

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Table 2.

Relationship between expression of stromal Cav-1 and HER-2/neu amplification.

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Figure 5.

CTCs stained with anti-CD45-CEP8-DAPI in peripheral blood of pancreatic cancer patients.

A: CTCs negative in patients with stromal Cav-1 loss. B: CTCs positive in patients with stromal Cav-1 loss. C: CTCs negative in patients with stromal Cav-1 expression. D: CTCs positive in patients with stromal Cav-1 expression. The white scale bar indicates 10 µm (x400).

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Figure 6.

Kaplan–Meier analysis of the overall postoperative survival curves in pancreatic cancer cases according to immunohistochemical staining as positive or negative of stromal Cav-1 expression.

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